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A potentially fatal complication of postoperative vomiting Boerhaave's syndrome

A potentially fatal complication of postoperative vomiting Boerhaave's syndrome Correspondence 257 on chronic diuretic therapy, the patient did not have tive team should be aware of this distressing com- any electrolyte disorder, which may predispose to plication, especially in patients with vitamin neurologic dysfunction. deficiency or neurological dysfunction. Long-term The patient was on chronic dexamethasone and patient follow-up is important to rule out latent or oestradiol therapy; and chronic steroid therapy may evolving neuropathy. be associated with neuropathy [6]. Although diffi- J. A. Alfa cult to prove, this may have been a contributory Department of Anaesthesiology factor to the myoclonus in this case report. Another 1H247 University Hospital possible contributory factor to the myoclonus in Ann Arbor this patient is the vitamin B deficiency that Michigan, USA was reported. Vitamin B deficiency is associated with myelopathy and neuropathy [7]. However, O. A. Bamgbade the degree of deficiency in our patient was mild Department of Anaesthesia and the patient did not require vitamin B therapy Central Manchester University Hospital for permanent resolution of myoclonus. Lower Manchester, UK limb neuropathy after spinal anaesthesia has been reported in a patient with thiamine deficiency [8], References but our patient had a normal thiamine level. 1. Cassim F, Houdayer E. Neurophysiology of myoclonus. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png European Journal of Anaesthesiology Wolters Kluwer Health

A potentially fatal complication of postoperative vomiting Boerhaave's syndrome

Samra, G. S.
European Journal of Anaesthesiology , Volume 25 (3) – Mar 1, 2008
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References (3)

Copyright
© 2008 Copyright European Society of Anaesthesiology
ISSN
0265-0215
eISSN
1365-2346
DOI
10.1017/S0265021507002773
pmid
18226284
Publisher site
See Article on Publisher Site

Abstract

Correspondence 257 on chronic diuretic therapy, the patient did not have tive team should be aware of this distressing com- any electrolyte disorder, which may predispose to plication, especially in patients with vitamin neurologic dysfunction. deficiency or neurological dysfunction. Long-term The patient was on chronic dexamethasone and patient follow-up is important to rule out latent or oestradiol therapy; and chronic steroid therapy may evolving neuropathy. be associated with neuropathy [6]. Although diffi- J. A. Alfa cult to prove, this may have been a contributory Department of Anaesthesiology factor to the myoclonus in this case report. Another 1H247 University Hospital possible contributory factor to the myoclonus in Ann Arbor this patient is the vitamin B deficiency that Michigan, USA was reported. Vitamin B deficiency is associated with myelopathy and neuropathy [7]. However, O. A. Bamgbade the degree of deficiency in our patient was mild Department of Anaesthesia and the patient did not require vitamin B therapy Central Manchester University Hospital for permanent resolution of myoclonus. Lower Manchester, UK limb neuropathy after spinal anaesthesia has been reported in a patient with thiamine deficiency [8], References but our patient had a normal thiamine level. 1. Cassim F, Houdayer E. Neurophysiology of myoclonus.

Journal

European Journal of Anaesthesiology – Wolters Kluwer Health

Published: Mar 1, 2008

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