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Abstracts

Abstracts Clinical Endocrinology (2016) 84 (Suppl. 1), 1–49 doi: 10.1111/cen.13010 4 20 Estrogen paradox: how can estrogen both cause and The vitamin D receptor (VDR) is expressed in skeletal prevent breast cancer? muscle of male mice and modulates 25-hydroxyvitamin D (25OHD) uptake in myofibers 1 1 1 Richard Santen , Wei Yue and Ji-Ping Wang Christian Girgis University of Virginia, Charlottesville, VA, United States Westmead Millennium Institute, Sydney, NSW, Australia Epidemiological studies provide strong evidence that estrogens con- tribute to the development of de novo breast cancerand to their growth For centuries, children with rickets were reported to demonstrate muscle when established. Specifically, these studies demonstrate an increased risk wasting, weakness and hypotonia. Similarly, adults with vitamin D defi- of breast cancer with early menarche, delayed first birth, late menopause, ciency commonly display myalgia, muscle weakness and type 2 muscle high body mass index, breast density, plasma estrogen levels, and use of fibre atrophy, which reverse with vitamin D supplementation. However, menopausal hormone therapy (MHT) containing estrogen plus a pro- vitamin D’s effects in muscle are elusive. Whether its effects in muscle gestogen. Paradoxically, estrogen therapy alone in the Women’s Health are predominantly indirect, via calcium and/or phosphate levels, or http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Clinical Endocrinology Wiley

Abstracts

Clinical Endocrinology , Volume 84 – Mar 1, 2016
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Publisher
Wiley
Copyright
Copyright © 2016 John Wiley & Sons Ltd
ISSN
0300-0664
eISSN
1365-2265
DOI
10.1111/cen.13010
pmid
26935124
Publisher site
See Article on Publisher Site

Abstract

Clinical Endocrinology (2016) 84 (Suppl. 1), 1–49 doi: 10.1111/cen.13010 4 20 Estrogen paradox: how can estrogen both cause and The vitamin D receptor (VDR) is expressed in skeletal prevent breast cancer? muscle of male mice and modulates 25-hydroxyvitamin D (25OHD) uptake in myofibers 1 1 1 Richard Santen , Wei Yue and Ji-Ping Wang Christian Girgis University of Virginia, Charlottesville, VA, United States Westmead Millennium Institute, Sydney, NSW, Australia Epidemiological studies provide strong evidence that estrogens con- tribute to the development of de novo breast cancerand to their growth For centuries, children with rickets were reported to demonstrate muscle when established. Specifically, these studies demonstrate an increased risk wasting, weakness and hypotonia. Similarly, adults with vitamin D defi- of breast cancer with early menarche, delayed first birth, late menopause, ciency commonly display myalgia, muscle weakness and type 2 muscle high body mass index, breast density, plasma estrogen levels, and use of fibre atrophy, which reverse with vitamin D supplementation. However, menopausal hormone therapy (MHT) containing estrogen plus a pro- vitamin D’s effects in muscle are elusive. Whether its effects in muscle gestogen. Paradoxically, estrogen therapy alone in the Women’s Health are predominantly indirect, via calcium and/or phosphate levels, or

Journal

Clinical Endocrinology – Wiley

Published: Mar 1, 2016

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