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Nuclear receptor ERRα and coactivator PGC-1β are effectors of IFN-γ-induced host defense

Nuclear receptor ERRα and coactivator PGC-1β are effectors of IFN-γ-induced host defense Downloaded from genesdev.cshlp.org on November 4, 2021 - Published by Cold Spring Harbor Laboratory Press Nuclear receptor ERR and coactivator PGC-1 are effectors of IFN--induced host defense 1 2,3,10 1,10 1,4 1 Junichiro Sonoda, Josée Laganière, Isaac R. Mehl, Grant D. Barish, Ling-Wa Chong, 5 6 7 8 8 Xiangli Li, Immo E. Scheffler, Dennis C. Mock, Alain R. Bataille, François Robert, 9 2,3,12 1,11 Chih-Hao Lee, Vincent Giguère, and Ronald M. Evans Howard Hughes Medical Institute and Gene Expression Laboratory, The Salk Institute for Biological Studies, La Jolla, California 92037, USA; Molecular Oncology Group, Departments of Medicine and Oncology, McGill University Health Centre, Montréal, Québec H3A 1A1, Canada; Department of Biochemistry, McGill University, Montréal, Québec H3G 1Y6, Canada; Division of Endocrinology and Metabolism, Department of Medicine, University of California, San Francisco, San Francisco, California 94121, USA; Department of Immunology, The Scripps Research Institute, La Jolla, California 92037, USA; Division of Biology, Molecular Biology Section, University of California, San Diego, La Jolla, California 92093 7 8 USA; San Diego Supercomputer Center, University of California, San Diego, La Jolla, California 92093, USA; Laboratory of Chromatin and Genomic Expression, Institut de Recherches Cliniques de Montréal, Montréal, Québec H2W 1R7, Canada; Harvard http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Genes & Development Unpaywall

Nuclear receptor ERRα and coactivator PGC-1β are effectors of IFN-γ-induced host defense

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Unpaywall
ISSN
0890-9369
DOI
10.1101/gad.1553007
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Abstract

Downloaded from genesdev.cshlp.org on November 4, 2021 - Published by Cold Spring Harbor Laboratory Press Nuclear receptor ERR and coactivator PGC-1 are effectors of IFN--induced host defense 1 2,3,10 1,10 1,4 1 Junichiro Sonoda, Josée Laganière, Isaac R. Mehl, Grant D. Barish, Ling-Wa Chong, 5 6 7 8 8 Xiangli Li, Immo E. Scheffler, Dennis C. Mock, Alain R. Bataille, François Robert, 9 2,3,12 1,11 Chih-Hao Lee, Vincent Giguère, and Ronald M. Evans Howard Hughes Medical Institute and Gene Expression Laboratory, The Salk Institute for Biological Studies, La Jolla, California 92037, USA; Molecular Oncology Group, Departments of Medicine and Oncology, McGill University Health Centre, Montréal, Québec H3A 1A1, Canada; Department of Biochemistry, McGill University, Montréal, Québec H3G 1Y6, Canada; Division of Endocrinology and Metabolism, Department of Medicine, University of California, San Francisco, San Francisco, California 94121, USA; Department of Immunology, The Scripps Research Institute, La Jolla, California 92037, USA; Division of Biology, Molecular Biology Section, University of California, San Diego, La Jolla, California 92093 7 8 USA; San Diego Supercomputer Center, University of California, San Diego, La Jolla, California 92093, USA; Laboratory of Chromatin and Genomic Expression, Institut de Recherches Cliniques de Montréal, Montréal, Québec H2W 1R7, Canada; Harvard

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Genes & Development – Unpaywall

Published: Aug 1, 2007

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