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Late Breaking Poster Session Group II – Red LB TPS 13

Late Breaking Poster Session Group II – Red LB TPS 13 Pniewska, E1; Kupry-Lipi ska, I2; Kuna, P2; Pawliczak, R1 s n Medical University of Lodz, Department of Immunopathology, Lodz, Poland; 2Medical University of Lodz, Department of Internal Diseases, Asthma and Allergy, Lodz, Poland matics and healthy subjects. cPLA2c may constitute a marker of asthma severity. 1. Moore WC, Meyers DA, Wenzel SE, Teague WG, Li H, Li X, et al. Identification of asthma phenotypes using cluster analysis in the Severe Asthma Research Program. Am J Respir Crit Care Med 2010, 181:315–323. Background: There are many phenotypes of bronchial asthma. Based on unsupervised hierarchical cluster analysis Moore et al.[1] distinguish five distinct clinical phenotypes of asthma. Asthma is characterized by chronic inflammation. Lipopolysaccharide and Der p1 participate in initiation and exacerbation of inflammatory process in asthmatics. Phospholipases A2 release the membrane unsaturated fatty acids that are directed to enzymes responsible for eicosanoids generation. These lipid mediators influence the course of inflammation in asthma. The aim of the study was to assess changes in phospholipase A2 expression in response to LPS and nDer p1 between severe, non-severe asthmatics and healthy subjects. Method: Twelve severe (IV, V cluster), 15 non-severe (I,II cluster) atopic to house dust mite asthmatics and 15 healthy http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Allergy Wiley

Late Breaking Poster Session Group II – Red LB TPS 13

Allergy , Volume 70 – Sep 1, 2015
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Publisher
Wiley
Copyright
Copyright © 2015 John Wiley & Sons A/S. Published by John Wiley & Sons Ltd
ISSN
0105-4538
eISSN
1398-9995
DOI
10.1111/all.12723
Publisher site
See Article on Publisher Site

Abstract

Pniewska, E1; Kupry-Lipi ska, I2; Kuna, P2; Pawliczak, R1 s n Medical University of Lodz, Department of Immunopathology, Lodz, Poland; 2Medical University of Lodz, Department of Internal Diseases, Asthma and Allergy, Lodz, Poland matics and healthy subjects. cPLA2c may constitute a marker of asthma severity. 1. Moore WC, Meyers DA, Wenzel SE, Teague WG, Li H, Li X, et al. Identification of asthma phenotypes using cluster analysis in the Severe Asthma Research Program. Am J Respir Crit Care Med 2010, 181:315–323. Background: There are many phenotypes of bronchial asthma. Based on unsupervised hierarchical cluster analysis Moore et al.[1] distinguish five distinct clinical phenotypes of asthma. Asthma is characterized by chronic inflammation. Lipopolysaccharide and Der p1 participate in initiation and exacerbation of inflammatory process in asthmatics. Phospholipases A2 release the membrane unsaturated fatty acids that are directed to enzymes responsible for eicosanoids generation. These lipid mediators influence the course of inflammation in asthma. The aim of the study was to assess changes in phospholipase A2 expression in response to LPS and nDer p1 between severe, non-severe asthmatics and healthy subjects. Method: Twelve severe (IV, V cluster), 15 non-severe (I,II cluster) atopic to house dust mite asthmatics and 15 healthy

Journal

Allergy – Wiley

Published: Sep 1, 2015

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