Renal infarction management: towards an etiological approach?

Renal infarction management: towards an etiological approach? Editorial Comment Renal infarction management: towards an etiological approach? a,b a b,c Jean-Philippe Lengele´ , Jean-Louis Christophe , and Alexandre Persu See original paper on page 634 n 1993, a 66-year-old woman was admitted to our and (iv) apparently idiopathic forms. Since then, all retro- Emergency Department for hypertensive crisis. She spective studies used this classification. In some publica- I complained of nausea, vomiting and abdominal pain. tions [3,4], the most frequent cause was the embolic form A biological work-up disclosed moderate renal impairment (50%), whereas other reported an equal distribution (25% (plasma creatinine: 1.7 mg/dl, eGFR: 32 ml/min per 1.73 m ) each) of embolic, vascular and idiopathic forms [2]. with neutrophilic leukocytosis (20.180/ml, 93% neutrophils) In the current issue of the Journal of Hypertension, and Lactate Deshydrogenase elevation (985 UI/l). Renal Faucon et al. [5] carefully reviewed 186 cases of renal infarction of the left kidney was demonstrated by computed infarction admitted in their tertiary center from July 2000 tomography-angiography (CTA). Arteriography highlighted to June 2015. They reported the causes of renal infarction a severe stenosis (90%) of the left renal artery with partial according to the four categories described previously [2]. In thrombosis of a poststenotic http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Hypertension Wolters Kluwer Health

Renal infarction management: towards an etiological approach?

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Publisher
Wolters Kluwer
Copyright
Copyright © 2018 Wolters Kluwer Health, Inc. All rights reserved.
ISSN
0263-6352
eISSN
1473-5598
D.O.I.
10.1097/HJH.0000000000001629
Publisher site
See Article on Publisher Site

Abstract

Editorial Comment Renal infarction management: towards an etiological approach? a,b a b,c Jean-Philippe Lengele´ , Jean-Louis Christophe , and Alexandre Persu See original paper on page 634 n 1993, a 66-year-old woman was admitted to our and (iv) apparently idiopathic forms. Since then, all retro- Emergency Department for hypertensive crisis. She spective studies used this classification. In some publica- I complained of nausea, vomiting and abdominal pain. tions [3,4], the most frequent cause was the embolic form A biological work-up disclosed moderate renal impairment (50%), whereas other reported an equal distribution (25% (plasma creatinine: 1.7 mg/dl, eGFR: 32 ml/min per 1.73 m ) each) of embolic, vascular and idiopathic forms [2]. with neutrophilic leukocytosis (20.180/ml, 93% neutrophils) In the current issue of the Journal of Hypertension, and Lactate Deshydrogenase elevation (985 UI/l). Renal Faucon et al. [5] carefully reviewed 186 cases of renal infarction of the left kidney was demonstrated by computed infarction admitted in their tertiary center from July 2000 tomography-angiography (CTA). Arteriography highlighted to June 2015. They reported the causes of renal infarction a severe stenosis (90%) of the left renal artery with partial according to the four categories described previously [2]. In thrombosis of a poststenotic

Journal

Journal of HypertensionWolters Kluwer Health

Published: Mar 1, 2018

References

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