Access the full text.
Sign up today, get DeepDyve free for 14 days.
P. Madsen, H. Rasmussen, Henrik Leffers, Bent Honoré, K. Dejgaard, E. Olsen, Jette Kiil, E. Walbum, A. Andersen, B. Basse, J. Lauridsen, G. Ratz, A. Celis, Joël Vandekerckhove, Julio Celis (1991)
Molecular cloning, occurrence, and expression of a novel partially secreted protein "psoriasin" that is highly up-regulated in psoriatic skin.The Journal of investigative dermatology, 97 4
Hofmann Bowman (2011)
92Am J Cardiovasc Dis, 1
B. Davidovici, N. Sattar, J. Prinz, L. Puig, P. Emery, J. Barker, P. Kerkhof, M. Ståhle, F. Nestle, G. Girolomoni, J. Krueger (2010)
Psoriasis and systemic inflammatory diseases: potential mechanistic links between skin disease and co-morbid conditions.The Journal of investigative dermatology, 130 7
M. Bowman, A. Schmidt (2011)
S100/calgranulins EN-RAGEing the blood vessels: implications for inflammatory responses and atherosclerosis.American journal of cardiovascular disease, 1 1
D. Gladman, Miriam Ang, Li Su, B. Tom, C. Schentag, V. Farewell (2008)
Cardiovascular morbidity in psoriatic arthritisAnnals of the Rheumatic Diseases, 68
W. Boehncke, S. Boehncke, A. Tobin, B. Kirby (2011)
The ‘psoriatic march’: a concept of how severe psoriasis may drive cardiovascular comorbidityExperimental Dermatology, 20
B. Enany, A. Zohiery, R. Elhilaly, T. Badr (2012)
Carotid intima-media thickness and serum leptin in psoriasisHerz, 37
Sadhanah Mazlan, Mohd Said, H. Hussein, Khadijah Shamsuddin, S. Shah, H. Basri (2009)
A study of intima media thickness and their cardiovascular risk factors in patients with psoriatic arthritis.Acta medica, 52 3
P. Rocha-Pereira, A. Santos-Silva, A. Santos-Silva, I. Rebelo, I. Rebelo, A. Figueiredo, A. Quintanilha, A. Quintanilha, F. Teixeira (2001)
Dislipidemia and oxidative stress in mild and in severe psoriasis as a risk for cardiovascular disease.Clinica chimica acta; international journal of clinical chemistry, 303 1-2
E. Shubbar, Jenny Vegfors, M. Carlström, S. Petersson, C. Enerbäck (2012)
Psoriasin (S100A7) increases the expression of ROS and VEGF and acts through RAGE to promote endothelial cell proliferationBreast Cancer Research and Treatment, 134
A. Batycka‐Baran, J. Maj, R. Wolf, J. Szepietowski (2014)
The New Insight into the Role of Antimicrobial Proteins-Alarmins in the Immunopathogenesis of PsoriasisJournal of Immunology Research, 2014
Dagmar Wilsmann-Theis, J. Wagenpfeil, Dirk Holzinger, Johannes Roth, Susanne Koch, S. Schnautz, T. Bieber, Joerg Wenzel (2016)
Among the S100 proteins, S100A12 is the most significant marker for psoriasis disease activityJournal of the European Academy of Dermatology and Venereology, 30
R. Eckert, A. Broome, Monica Ruse, N. Robinson, D. Ryan, Kathleen Lee (2004)
S100 proteins in the epidermis.The Journal of investigative dermatology, 123 1
Salama (2013)
33Dis Markers, 34
F. Späh (2008)
Inflammation in atherosclerosis and psoriasis: common pathogenic mechanisms and the potential for an integrated treatment approachBritish Journal of Dermatology, 159
Y. Liu, J. Krueger, A. Bowcock (2007)
Psoriasis: genetic associations and immune system changesGenes and Immunity, 8
A. Batycka‐Baran, A. Batycka‐Baran, E. Hattinger, S. Zwicker, B. Summer, O. Howard, Peter Thomas, J. Szepietowski, T. Ruzicka, J. Prinz, R. Wolf (2015)
Leukocyte-derived koebnerisin (S100A15) and psoriasin (S100A7) are systemic mediators of inflammation in psoriasis.Journal of dermatological science, 79 3
S. El-Mongy, H. Fathy, A. Abdelaziz, E. Omran, S. George, N. Neseem, N. EL-NOUR (2009)
Subclinical atherosclerosis in patients with chronic psoriasis: a potential associationJournal of the European Academy of Dermatology and Venereology, 24
G. Kapuku, G. Harshfield, H. Davis, F. Treiber (2006)
Early markers of cardiovascular disease.Vascular pharmacology, 45 5
R. Salama, H. Al‐Shobaili, Ahmad Robaee, A. Alzolibani (2012)
Psoriasin: A Novel Marker Linked Obesity with PsoriasisDisease markers, 34
R. Donato (2003)
Intracellular and extracellular roles of S100 proteinsMicroscopy Research and Technique, 60
R. Wolf, A. Mirmohammadsadegh, M. Walz, B. Lysa, U. Tartler, R. Remus, U. Hengge, G. Michel, T. Ruzicka (2003)
Molecular cloning and characterization of alternatively spliced mRNA isoforms from psoriatic skin encoding a novel member of the S100 familyThe FASEB Journal, 17
S. Miyamoto, M. Ueda, Masaki Ikemoto, T. Naruko, Akira Itoh, Shunichi Tamaki, Ryuji Nohara, Fumio Terasaki, S. Sasayama, Masatoshi Fujita (2008)
Increased serum levels and expression of S100A8/A9 complex in infiltrated neutrophils in atherosclerotic plaque of unstable anginaHeart, 94
V. Antonucci, V. Tengattini, Riccardo Balestri, A. Patrizi, M. Filippini, F. Bardazzi (2014)
Intima‐media thickness in an Italian psoriatic population: correlation with lipidic serum levels, PASI and BMIJournal of the European Academy of Dermatology and Venereology, 28
E. Altekin, Serkan Koç, M. Karakaş, A. Yanıkoğlu, I. Başarıcı, I. Demir, E. Alpsoy (2012)
Determination of subclinical atherosclerosis in plaque type psoriasis patients without traditional risk factors for atherosclerosis.Turk Kardiyoloji Dernegi arsivi : Turk Kardiyoloji Derneginin yayin organidir, 40 7
A. Karadağ, B. Yavuz, D. Ertuğrul, K. Akın, A. Yalçın, O. Deveci, N. Ata, M. Kucukazman, K. Dal (2010)
Is psoriasis a pre‐atherosclerotic disease? Increased insulin resistance and impaired endothelial function in patients with psoriasisInternational Journal of Dermatology, 49
IntroductionPsoriasis is an immune‐mediated inflammatory disease that affects approximately 1–3% of the population worldwide. An increasing body of evidence supports that the inflammatory process in psoriasis may have systemic consequences with implications for the development of psoriatic comorbidities, including cardiovascular disease (CVD). Psoriasis was suggested to be an independent risk factor for atherosclerosis and CVD.Current concepts favour the idea that inflammation provides a pivotal link between psoriasis and atherosclerosis, which seems to be responsible for the association between the two diseases. Overall, many of the inflammatory products produced in psoriatic skin lesions appear to be released into the systemic circulation as a function of the severity and extent of skin lesions. During periods of active disease, skin‐derived inflammatory products could alter the properties of leucocytes while circulating through the inflamed cutaneous vasculature, or affect endothelial cells at distant sites.The S100 proteins constitute a family of small calcium‐binding proteins that are increasingly recognised as potential players in the pathogenesis of various inflammatory diseases. Several lines of evidence support a crucial role of S100 proteins in the pathogenesis of atherosclerosis, and several S100 molecules have been found to be upregulated in psoriasis, implicating their involvement in its pathogenesis.The antimicrobial S100A proteins
Clinical & Experimental Dermatology – Wiley
Published: Jan 1, 2018
Read and print from thousands of top scholarly journals.
Already have an account? Log in
Bookmark this article. You can see your Bookmarks on your DeepDyve Library.
To save an article, log in first, or sign up for a DeepDyve account if you don’t already have one.
Copy and paste the desired citation format or use the link below to download a file formatted for EndNote
Access the full text.
Sign up today, get DeepDyve free for 14 days.
All DeepDyve websites use cookies to improve your online experience. They were placed on your computer when you launched this website. You can change your cookie settings through your browser.