Letters to the Editor In conclusion, there may be variations in the clinical presen- REFERENCES tation, laboratory ﬁndings and response to treatment of AE. 1 Zhou XY, Chen XJ, Wang S et al. One recurrent homozygous muta- Therefore, we suggest that the diagnosis should be conﬁrmed tion of SLC39A4 in a girl with acrodermatitis enteropathica from with the mutation analysis of the SLC39A4 gene. southwestern China. Int J Dermatol. 2016; 55: 223–225. 2Kur € y S, Kharﬁ M, Kamoun R et al. Mutation spectrum of human SLC39A4 in a panel of patients with acrodermatitis enteropathica. CONFLICT OF INTEREST: None declared. Hum Mutat 2003; 22: 337–338. 3 Kilic M, Taskesen M, Coskun T et al. A zinc sulphate-resistant acro- 1 1 dermatitis enteropathica patient with a novel mutation in SLC39A4 Hilal KAYA ERDOGAN, Isil BULUR, gene. JIMD Rep 2012; 2:25–28. 1 2 Zeynep Nurhan SARACOGLU, Huseyin ASLAN, 4 Kilic SS, Giraud M, Schmitt S et al. A novel mutation of the SLC39A4 3 1 gene causing acrodermatitis enteropathica. Br J Dermatol 2007; 157: Sultan Durmus AYDOGDU, Bahadir YILDIZ 1 2 3 386–387. Departments of Dermatology, Genetics, and Pediatrics, Faculty of 5 Garza-Rodrı´guez V, de la Fuente-Garcıa A, Liy-Wong C et
The Journal of Dermatology – Wiley
Published: Aug 1, 2016
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