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Editorial important contributions to our understanding of these processes. The manner in which ‘organizers’ influence embryological differentiation should also offer a fruitful field of investigation, which has as yet hardly been explored. An understanding of normal regulatory mechanisms and feed-back systems is a prerequisite to an understanding of those failures in control of tissue proliferation that result in neoplasia. The applications of kinetic studies to the cancer problem are numerous. The growth rate of a tumour as a whole is determined by a balance between the rates of cell proliferation and of cell death. Recent work has brought to light the fact that in some comparatively slowly-growing tumours, notably certain lymphomas, cellular turnover may be relatively rapid. This type of observation stresses the influences that host factors may exert on tumour growth, revives the concept that immunological phenomena may play a part in the regulation of cellular proliferation and helps to explain the sensitivity of these tumours to radiation and to drugs. Accurate determination of cell cycle kinetics may aid in the selection and phasing of therapeutic agents, for achieving the greatest differential cytotoxic effect on tumour cells in relation to that on normal tissues. This brief survey http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Cell Proliferation Wiley

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