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; obesity; eride metabolism obese ; muscle; triglyc- GREENWOOD HAS SUGGESTED (l&16) that the primary event the development of some forms of obesity is not hyperphagia but rather abnormal partitiong of fuel toward storage adipose tissue relative to muscle, with hyperphagia a secondary result of this defect. She hypothesized that abnormalities adipose tissue (ATLPL) might play a central role this process. Evidence support of this hypothesis came from measurements of ATLPL activity lean, obese, and obese pair fed to lean controls (6). This study showed creased ATLPL obese relative to lean controls and no change or a mild crease pairfed . Pair-fed weighed significantly less than ad libitum-fed obese controls, but body composition, particular percent body fat, was not changed by caloric restriction. This study was done, however, without controllg for feedg effects at the time of death, and the mechanisms responsible for the crease enzyme activity were not addressed. obese humans who successfully achieve a significant E246 0193-1849/91 $1.50 Copyright reduction weight, failure to mata the weight loss is a frustratg and all too common problem. Reducedobesity is a condition where mechanisms present durg the origal development of obesity might be reexpressed. A number of studies have
AJP - Endocrinology and Metabolism – The American Physiological Society
Published: Aug 1, 1991
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