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~., D. FRANKL, J. , JR. Hyperfunction with normal otropic state of the hypertrophied ventricle. Am. J. Physiol. 232(4): H41tLH425, 1977 or Am. J. Physiol.: Heart Circ. Physiol. l(4): H4WH425, 1977. -Conscious dogs were strumented with an flatable cuff around the ascendg aorta, a high-fidelity micromanometer the ventricle (LV), pairs of ultrasonic crystals for measurements of LV wall thickness ternal LV diameter. Wall stress (WSt) mean velocity of wall shorteng (Vc,) were calculated. Mean force-velocity relations WSt-diameter loops sgle contractions were then analyzed over a range of matched systolic pressures durg acute aortic constrictions both before after duction of chronic by sustaed aortic constriction. At normal LV systolic pressures at each matched level of systolic LV pressure, wall shorteng velocity was creased the hypertrophied ventricle. However, force-velocity relations obtaed by relatg mean VCF to mean WSt at various stress levels fell on the same relation as durg control. The lear relation between LV diameter pressure at the end of was shifted to the the hypertrophied ventricle, dicatg enhanced shorteng. However, lear WSt-diameter relations at end- were not different control hypertrophied hearts. These fdgs dicate that the ventricle hypertrophied by pressure overload exhibited hyperfunction as a pump but that
AJP - Heart and Circulatory Physiology – The American Physiological Society
Published: Apr 1, 1977
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