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Effects of chest compression on reflex ventilatory drive and pulmonary function

Effects of chest compression on reflex ventilatory drive and pulmonary function Abstract External compression of the chest sufficient to reduce the lung volume (FRC) by 1 liter in eight normal subjects interfered with the mechanical function of the lungs. We have confirmed the findings of Caro et al. ( J. Clin. Invest. 39: 573, 1960), who showed a decrease in lung compliance and an increase in respiratory rate. Neither returned to normal when the compressing force was removed, and it was not until the subject took a deep breath that the lungs returned to their control state. We also found a reduction in anatomical dead space and alveolar hyperventilation. Arterial blood gas tensions showed evidence of complex ventilation-perfusion abnormalities, which could not be explained by any single factor. We think the hyperventilation associated with chest compression is reflex in origin and related to a decrease in lung volume rather than to any change in transpulmonary pressure. Submitted on January 4, 1962 Copyright © 1962 the American Physiological Society http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Applied Physiology The American Physiological Society

Effects of chest compression on reflex ventilatory drive and pulmonary function

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Publisher
The American Physiological Society
Copyright
Copyright © 1962 the American Physiological Society
ISSN
8750-7587
eISSN
1522-1601
Publisher site
See Article on Publisher Site

Abstract

Abstract External compression of the chest sufficient to reduce the lung volume (FRC) by 1 liter in eight normal subjects interfered with the mechanical function of the lungs. We have confirmed the findings of Caro et al. ( J. Clin. Invest. 39: 573, 1960), who showed a decrease in lung compliance and an increase in respiratory rate. Neither returned to normal when the compressing force was removed, and it was not until the subject took a deep breath that the lungs returned to their control state. We also found a reduction in anatomical dead space and alveolar hyperventilation. Arterial blood gas tensions showed evidence of complex ventilation-perfusion abnormalities, which could not be explained by any single factor. We think the hyperventilation associated with chest compression is reflex in origin and related to a decrease in lung volume rather than to any change in transpulmonary pressure. Submitted on January 4, 1962 Copyright © 1962 the American Physiological Society

Journal

Journal of Applied PhysiologyThe American Physiological Society

Published: Jul 1, 1962

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