Cardiovascular parameters in emphysematous and control horses

Cardiovascular parameters in emphysematous and control horses EBERLY, V. E., W, S. AND J. R. GILLESPIE. Cardz’ovascular parameters in and control . J. Appl. 1966. - with clinical signs Physiol. 2 I (3) : 883-889. chronic pulmonary emphysema were demonstrated to have heart rates, total peripheral vascular resistance, pulmonary vascular resistance, and mean pulmonary artery blood pressure highly significantly greater than normal. They also had a highly significant decrease in central blood volume, stroke volume, stroke index, and left ventricular \dTork. Cardiac out- put was significantly lower than in control (normal) . There were no significant differences in arterial or right vengroup had variable tricular blood pressures. The diseased values, probably depending on the degree and extent pul- monary pathology cardiac ; present. emphysema; cardiopulmonary physiology large air spaces, with loss alveolar tissue. Chronic or infectious bronchitis may also be present. The present study was designed to determine the differences in parameters between control and with long-standing signs heaves. Spiirri and Schlatter (20) reported greater pulmonary artery pressures and amplitudes in three with reported postmortem clinical signs heaves. They findings right ventricular hypertrophy, pulmonary emphysema, and bronchiolitis in two the cases; the third was not observed. Grover (2) found a greater pulmonary artery pressure in http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Applied Physiology The American Physiological Society

Cardiovascular parameters in emphysematous and control horses

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Publisher
The American Physiological Society
Copyright
Copyright © 1966 the American Physiological Society
ISSN
8750-7587
eISSN
1522-1601
Publisher site
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Abstract

EBERLY, V. E., W, S. AND J. R. GILLESPIE. Cardz’ovascular parameters in and control . J. Appl. 1966. - with clinical signs Physiol. 2 I (3) : 883-889. chronic pulmonary emphysema were demonstrated to have heart rates, total peripheral vascular resistance, pulmonary vascular resistance, and mean pulmonary artery blood pressure highly significantly greater than normal. They also had a highly significant decrease in central blood volume, stroke volume, stroke index, and left ventricular \dTork. Cardiac out- put was significantly lower than in control (normal) . There were no significant differences in arterial or right vengroup had variable tricular blood pressures. The diseased values, probably depending on the degree and extent pul- monary pathology cardiac ; present. emphysema; cardiopulmonary physiology large air spaces, with loss alveolar tissue. Chronic or infectious bronchitis may also be present. The present study was designed to determine the differences in parameters between control and with long-standing signs heaves. Spiirri and Schlatter (20) reported greater pulmonary artery pressures and amplitudes in three with reported postmortem clinical signs heaves. They findings right ventricular hypertrophy, pulmonary emphysema, and bronchiolitis in two the cases; the third was not observed. Grover (2) found a greater pulmonary artery pressure in

Journal

Journal of Applied PhysiologyThe American Physiological Society

Published: May 1, 1966

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