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rats CYNTHIA GWILLIAM Ottawa, Ontario, Canada KIN 9A9 HIMMS-HAGEN,JEAN,JOANTRIAFILLOU,CYNTHIA GWILLXAM. of eteria-fed ruts. Am. J. Phhvsiol. (Endocrinol. 241 Metab. 4): El&E120, 1981.-FeedingU a âeteriaâ for 2 wk to male Holtzman rats resulted in a weight gain that was, on average, only slightly more than that of trol rats fed a regular chow . Wet weight, DNA, total protein tent of interscapular were more than doubled in the eteria-fed rats proliferation of mitochondria paralleled growth. After 2 wk of recovery from eteria feeding, the exped size of the had completely regressed to a normal level. mitochondria of eteria-fed rats bound 3 times more purine nucleotides than mitochondria of chow-fed trol rats, but no change in the pxoportion of polypeptides with molecular weight in the region of 32,000 could be detected. The changes in its mitochondria in eteria-fed rats correspond to those seen previously in noradrenalinetreated rats, i.e., growth accompanied by mitochondrial proliferation an unmasking of proton ductance pathways. The increase in 32,000-mol-wt polypeptides seen in mitochondria of cold-acclimated rats does not occur in the eteria-fed rats. trol mechanisms are presumed to differ, either quantitatively or qualitatively, in the two situations, cold exposure overeating, which both cause growth of .
AJP - Endocrinology and Metabolism – The American Physiological Society
Published: Aug 1, 1981
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