Adrenergic control of renin in euhydrated and water-deprived conscious dogs

Adrenergic control of renin in euhydrated and water-deprived conscious dogs 0193-1849/t% $1.50 cular tone (2, 15, 17) or sodium chloride transport at the macula densa region (19) that are not evident from measurements total renal blood flow or function. Although renal a-adrenoceptors have been demonstrated to contribute to reflex neural stimulation release anesthetized, surgically prepared experimental animals (1, 22, 26), the extent their contribution to plasma activity (PRA) animals is not fully understood. , renal arterial fusion a-adrenoceptor agonists creases PRA even at low agonist concentrations that have little or no effect on total renal vascular resistance (3), and reflex neural stimulation renal ac-adrenoceptors can modulate the relationship between secretion rate and renal perfusion pressure (15). These observations suggest that renal a-adrenoceptors may partially mediate creased PRA durg episodes elevated renal nerve activity unanesthetized normotensive animals. the present study, we therefore compared the effects on PRA renal arterial fusion the a-adrenoceptor antagonist phenoxybenzame and the ,&adrenoceptor antagonist propranolol sodium-replete studied two states (orthostatic stress alone and combation with 24-h water deprivation) which PRA was elevated to two- and threefold basal levels. We also evaluated the effects on PRA combed renal arterial fusion cyand ,&adrenoceptor antagonists order to quantify the total contribution stimulation to PRA both states. Fally, we http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png AJP - Endocrinology and Metabolism The American Physiological Society

Adrenergic control of renin in euhydrated and water-deprived conscious dogs

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Publisher
The American Physiological Society
Copyright
Copyright © 1988 the American Physiological Society
ISSN
0193-1849
eISSN
1522-1555
Publisher site
See Article on Publisher Site

Abstract

0193-1849/t% $1.50 cular tone (2, 15, 17) or sodium chloride transport at the macula densa region (19) that are not evident from measurements total renal blood flow or function. Although renal a-adrenoceptors have been demonstrated to contribute to reflex neural stimulation release anesthetized, surgically prepared experimental animals (1, 22, 26), the extent their contribution to plasma activity (PRA) animals is not fully understood. , renal arterial fusion a-adrenoceptor agonists creases PRA even at low agonist concentrations that have little or no effect on total renal vascular resistance (3), and reflex neural stimulation renal ac-adrenoceptors can modulate the relationship between secretion rate and renal perfusion pressure (15). These observations suggest that renal a-adrenoceptors may partially mediate creased PRA durg episodes elevated renal nerve activity unanesthetized normotensive animals. the present study, we therefore compared the effects on PRA renal arterial fusion the a-adrenoceptor antagonist phenoxybenzame and the ,&adrenoceptor antagonist propranolol sodium-replete studied two states (orthostatic stress alone and combation with 24-h water deprivation) which PRA was elevated to two- and threefold basal levels. We also evaluated the effects on PRA combed renal arterial fusion cyand ,&adrenoceptor antagonists order to quantify the total contribution stimulation to PRA both states. Fally, we

Journal

AJP - Endocrinology and MetabolismThe American Physiological Society

Published: Dec 1, 1988

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