The Protein Modification and Degradation Pathways after Brain Ischemia

The Protein Modification and Degradation Pathways after Brain Ischemia Transl. Stroke Res. (2018) 9:199–200 DOI 10.1007/s12975-017-0573-z EDITORIAL The Protein Modification and Degradation Pathways after Brain Ischemia 1,2 1,2 Tibor Kristian & Bingren Hu Received: 19 September 2017 /Accepted: 21 September 2017 /Published online: 1 October 2017 Springer Science+Business Media, LLC 2017 This Special Issue of Translational Stroke Research is focused of cellular metabolic pathways, some may have a more sig- on the protein modification and degradation pathways after nificant impact on cell fate than others. To survive this patho- brain ischemia and is a continuation of a previous special issue logic environment, post-ischemic neurons must cope with re- in this journal entitled: BThe protein degradation pathways pair and eliminate irreversibly damaged organelles and toxic materials by upregulation of cellular defense systems. after brain ischemia^ [1]. Articles in this current issue sum- marize recent developments and understanding of protein ag- This special issue of Translational Stroke Research is fo- gregation, modification, and degradation triggered by ische- cused on alterations in protein metabolism, degradation mech- mic insult. This Special Issue seeks to reiterate that disruption anisms, and post-translational modifications following brain in cellular protein metabolism remains an important aspect of ischemic insult. Each individual article highlights and dis- ischemic brain http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Translational Stroke Research Springer Journals

The Protein Modification and Degradation Pathways after Brain Ischemia

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Publisher
Springer US
Copyright
Copyright © 2017 by Springer Science+Business Media, LLC
Subject
Biomedicine; Neurosciences; Neurology; Cardiology; Neurosurgery; Vascular Surgery
ISSN
1868-4483
eISSN
1868-601X
D.O.I.
10.1007/s12975-017-0573-z
Publisher site
See Article on Publisher Site

Abstract

Transl. Stroke Res. (2018) 9:199–200 DOI 10.1007/s12975-017-0573-z EDITORIAL The Protein Modification and Degradation Pathways after Brain Ischemia 1,2 1,2 Tibor Kristian & Bingren Hu Received: 19 September 2017 /Accepted: 21 September 2017 /Published online: 1 October 2017 Springer Science+Business Media, LLC 2017 This Special Issue of Translational Stroke Research is focused of cellular metabolic pathways, some may have a more sig- on the protein modification and degradation pathways after nificant impact on cell fate than others. To survive this patho- brain ischemia and is a continuation of a previous special issue logic environment, post-ischemic neurons must cope with re- in this journal entitled: BThe protein degradation pathways pair and eliminate irreversibly damaged organelles and toxic materials by upregulation of cellular defense systems. after brain ischemia^ [1]. Articles in this current issue sum- marize recent developments and understanding of protein ag- This special issue of Translational Stroke Research is fo- gregation, modification, and degradation triggered by ische- cused on alterations in protein metabolism, degradation mech- mic insult. This Special Issue seeks to reiterate that disruption anisms, and post-translational modifications following brain in cellular protein metabolism remains an important aspect of ischemic insult. Each individual article highlights and dis- ischemic brain

Journal

Translational Stroke ResearchSpringer Journals

Published: Oct 1, 2017

References

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