The PPR protein SLOW GROWTH 4 is involved in editing of nad4 and affects the splicing of nad2 intron 1

The PPR protein SLOW GROWTH 4 is involved in editing of nad4 and affects the splicing of nad2... Plant Mol Biol (2017) 93:355–368 DOI 10.1007/s11103-016-0566-4 The PPR protein SLOW GROWTH 4 is involved in editing of nad4 and affects the splicing of nad2 intron 1 1 1,2 1 Stefan Weißenberger  · Jürgen Soll  · Chris Carrie   Received: 26 September 2016 / Accepted: 21 November 2016 / Published online: 9 December 2016 © Springer Science+Business Media Dordrecht 2016 Abstract another complex I subunit, was also decreased. The change Key message SLO4 is a mitochondrial PPR protein in splicing efficiency could however not be linked to any that is involved in editing nad4, possibly required for editing defects in the nad2 transcript. the efficient splicing of nad2 intron1. Abstract Pentatricopeptide repeat (PPR) proteins con- Keywords Mitochondria · Complex I · RNA editing · stitute a large protein family in flowering plants and are Splicing · Pentatricopeptide repeat thought to be mostly involved in organellar RNA metabo- lism. The subgroup of PLS-type PPR proteins were found to be the main specificity factors of cytidine to uridine Introduction RNA editing. Identifying the targets of PLS-type PPR proteins can help in elucidating the molecular function of Mitochondria play an important role in a multitude of pro- proteins encoded in the organellar genomes. In this study, cesses within eukaryotic cells. Although they harbor their plants http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Plant Molecular Biology Springer Journals

The PPR protein SLOW GROWTH 4 is involved in editing of nad4 and affects the splicing of nad2 intron 1

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Publisher
Springer Netherlands
Copyright
Copyright © 2016 by Springer Science+Business Media Dordrecht
Subject
Life Sciences; Plant Sciences; Biochemistry, general; Plant Pathology
ISSN
0167-4412
eISSN
1573-5028
D.O.I.
10.1007/s11103-016-0566-4
Publisher site
See Article on Publisher Site

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