The mouse homologs of RELA and MLK3 are located within a 120-kb fragment on Chromosome 19

The mouse homologs of RELA and MLK3 are located within a 120-kb fragment on Chromosome 19 Mammalian Genome 8,513-515 (1997). e//o///e 9 Springer-Verlag New York Inc. 1997 The mouse homologs of RELA and MLK3 are located within a 120-kb fragment on Chromosome 19 M. Fernandes, a C. Poirier, 2 N.J. Lassam, 3 C. Larsson, 4 J.-L. Gu~net, 2 P. Gaudray, 1 G.F. Carle 1 1CNRS/UNSA UMR 6549, Facult6 de Mddecine, F-06107 Nice, France 2Unite de Gtndtique des Mammif~res, Institut Pasteur de Paris, F-75724 Paris cedex 15, France 3Department of Medicine, University of Toronto, Toronto, Ontario, M5S 1AS, Canada 4Department of Medicine and Department of Molecular Medicine, Karolinska Hospital, L6 Building, S-171 76 Stockholm, Sweden Received: 29 January 1997 / Accepted: 19 March 1997 Recent advances in the identification of a large collection of poly- dependent diabetes mellitus type 4 (Hashimoto et al. 1994), Best's morphic genetic markers and unique probes have yielded respec- disease (Stone et al. 1992), Bardet-Biedl syndrome type 1 (Leppert tively detailed genetic and physical maps of both human and et al. 1994), and spinocerebellar ataxia type 5 (Ranum et al. 1994), mouse chromosomes. Since only a subset of these markers, that is, have also been assigned to this chromosomal region. genes, are useful for comparative mapping, the integration of Similarly, the http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Mammalian Genome Springer Journals

The mouse homologs of RELA and MLK3 are located within a 120-kb fragment on Chromosome 19

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Publisher
Springer-Verlag
Copyright
Copyright © 1997 by Springer-Verlag
Subject
Life Sciences; Cell Biology; Anatomy; Zoology
ISSN
0938-8990
eISSN
1432-1777
D.O.I.
10.1007/s003359900487
Publisher site
See Article on Publisher Site

Abstract

Mammalian Genome 8,513-515 (1997). e//o///e 9 Springer-Verlag New York Inc. 1997 The mouse homologs of RELA and MLK3 are located within a 120-kb fragment on Chromosome 19 M. Fernandes, a C. Poirier, 2 N.J. Lassam, 3 C. Larsson, 4 J.-L. Gu~net, 2 P. Gaudray, 1 G.F. Carle 1 1CNRS/UNSA UMR 6549, Facult6 de Mddecine, F-06107 Nice, France 2Unite de Gtndtique des Mammif~res, Institut Pasteur de Paris, F-75724 Paris cedex 15, France 3Department of Medicine, University of Toronto, Toronto, Ontario, M5S 1AS, Canada 4Department of Medicine and Department of Molecular Medicine, Karolinska Hospital, L6 Building, S-171 76 Stockholm, Sweden Received: 29 January 1997 / Accepted: 19 March 1997 Recent advances in the identification of a large collection of poly- dependent diabetes mellitus type 4 (Hashimoto et al. 1994), Best's morphic genetic markers and unique probes have yielded respec- disease (Stone et al. 1992), Bardet-Biedl syndrome type 1 (Leppert tively detailed genetic and physical maps of both human and et al. 1994), and spinocerebellar ataxia type 5 (Ranum et al. 1994), mouse chromosomes. Since only a subset of these markers, that is, have also been assigned to this chromosomal region. genes, are useful for comparative mapping, the integration of Similarly, the

Journal

Mammalian GenomeSpringer Journals

Published: Mar 24, 2009

References

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