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F. Kunst, N. Ogasawara, I. Moszer, A. Albertini, G. Alloni, V. Azevedo, M. Bertero, M. Bertero, P. Bessières, A. Bolotin, S. Borchert, R. Borriss, L. Boursier, A. Brans, M. Braun, S. Brignell, S. Bron, S. Brouillet, S. Brouillet, C. Bruschi, B. Caldwell, V. Capuano, N. Carter, S-K. Choi, Jean-Jacques Codani, I. Connerton, N. Cummings, R. Daniel, F. Denizot, K. Devine, A. Düsterhöft, S. Ehrlich, P. Emmerson, K. Entian, J. Errington, C. Fabret, E. Ferrari, D. Foulger, C. Fritz, M. Fujita, Y. Fujita, S. Fuma, A. Galizzi, N. Galleron, S. Ghim, P. Glaser, A. Goffeau, E. Golightly, G. Grandi, G. Guiseppi, B. Guy, K. Haga, J. Haiech, C. Harwood, A. Hénaut, H. Hilbert, S. Holsappel, S. Hosono, M. Hullo, M. Itaya, Louis Jones, B. Joris, D. Karamata, Y. Kasahara, M. Klaerr-Blanchard, C. Klein, Y. Kobayashi, P. Koetter, G. Koningstein, Susanne Krogh, M. Kumano, K. Kurita, A. Lapidus, S. Lardinois, J. Lauber, V. Lazarevic, S. Lee, A. Lévine, H. Liu, S. Masuda, C. Mauël, C. Médigue, C. Médigue, N. Medina, R. Mellado, M. Mizuno, D. Moestl, S. Nakai, M. Noback, D. Noone, M. O'Reilly, K. Ogawa, A. Ogiwara, B. Oudega, S. Park, V. Parro, T. Pohl, D. Portetelle, S. Porwollik, A. Prescott, E. Presecan, P. Pujić, B. Purnelle, G. Rapoport, M. Rey, Stacey Reynolds, M. Rieger, C. Rivolta, E. Rocha, E. Rocha, B. Roche, M. Rose, Y. Sadaie, Toshitada Sato, E. Scanlan, S. Schleich, R. Schroeter, F. Scoffone, J. Sekiguchi, A. Sękowska, S. Séror, P. Serror, B. Shin, B. Soldo, A. Sorokin, E. Tacconi, T. Takagi, H. Takahashi, K. Takemaru, M. Takeuchi, A. Tamakoshi, T. Tanaka, P. Terpstra, A. Tognoni, V. Tosato, S. Uchiyama, M. Vandenbol, F. Vannier, A. Vassarotti, A. Viari, R. Wambutt, E. Wedler, H. Wedler, T. Weitzenegger, P. Winters, A. Wipat, Hiroki Yamamoto, K. Yamane, K. Yasumoto, Katsunori Yata, K. Yoshida, H. Yoshikawa, E. Zumstein, H. Yoshikawa, Antoine Danchin (1997)
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Analysis of the phenotypic manifestation of inactivation of several genes from the ytlI–ytnM operon containing the ribR gene and results of Northern hybridization showed that theribR gene does not have the self promoter and is transcribed from the main promoter of the ytlI–ytnM operon. Two sites of single nucleotide substitutions leading to derepression of ribR gene were identified between the putative main promoter and transcription start of the ytlI–ytnM operon regulatory region.
Russian Journal of Genetics – Springer Journals
Published: Oct 18, 2004
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