Stress, chemocommunication, and the physiological hypothesis of mutation

Stress, chemocommunication, and the physiological hypothesis of mutation The review considers stress as a physiological state of the organism, affecting the cellular, genomic, and population levels. Literature data and cytogenetic studies by the author support basic statements of the physiological hypothesis of mutation, which was advanced as early as in the 1940s. Studies of pheromonal effects in germline and somatic cells of the house mouse demonstrated the role of olfactory stressors in generating genetic variation in microevolutionary changes. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Russian Journal of Genetics Springer Journals

Stress, chemocommunication, and the physiological hypothesis of mutation

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Publisher
Nauka/Interperiodica
Copyright
Copyright © 2007 by Pleiades Publishing, Inc.
Subject
Biomedicine; Human Genetics; Animal Genetics and Genomics; Microbial Genetics and Genomics
ISSN
1022-7954
eISSN
1608-3369
D.O.I.
10.1134/S102279540710002X
Publisher site
See Article on Publisher Site

Abstract

The review considers stress as a physiological state of the organism, affecting the cellular, genomic, and population levels. Literature data and cytogenetic studies by the author support basic statements of the physiological hypothesis of mutation, which was advanced as early as in the 1940s. Studies of pheromonal effects in germline and somatic cells of the house mouse demonstrated the role of olfactory stressors in generating genetic variation in microevolutionary changes.

Journal

Russian Journal of GeneticsSpringer Journals

Published: Oct 15, 2007

References

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