Regadenoson-induced hyperemia for absolute myocardial blood flow quantitation by 13N-ammonia PET and detection of cardiac allograft vasculopathy

Regadenoson-induced hyperemia for absolute myocardial blood flow quantitation by 13N-ammonia PET... EDITORIAL Regadenoson-induced hyperemia for absolute myocardial blood flow quantitation by N- ammonia PET and detection of cardiac allograft vasculopathy a,b,c Rene R. Sevag Packard, MD, PhD, and Jamshid Maddahi, MD, FACC, a,b,d FASNC Division of Cardiology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA Ronald Reagan UCLA Medical Center, Los Angeles, CA Veterans Affairs West Los Angeles Medical Center, Los Angeles, CA Nuclear Medicine Clinic, Department of Molecular and Medical Pharmacology, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA Received Oct 4, 2016; accepted Oct 5, 2016 doi:10.1007/s12350-016-0763-1 available organs has led to the rise of ventricular assist See related article, pp. 1134–1144 devices implanted as destination therapy and to a very rigorous transplant recipient selection process. Fol- lowing OHT, transplant recipients are exposed to a In this issue of the Journal of Nuclear Cardiology, stringent surveillance program periodically screening for Miguel H. Pampolini and associates report on non-in- graft rejection, graft failure, infection, malignancy, and vasive quantitation of myocardial blood flow (MBF) by other complications. CAV, or transplant vasculopathy, regadenoson positron emission tomography (PET) accounts for *10% of deaths in OHT recipients starting imaging for http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Nuclear Cardiology Springer Journals

Regadenoson-induced hyperemia for absolute myocardial blood flow quantitation by 13N-ammonia PET and detection of cardiac allograft vasculopathy

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Publisher
Springer US
Copyright
Copyright © 2017 by American Society of Nuclear Cardiology
Subject
Medicine & Public Health; Cardiology; Nuclear Medicine; Imaging / Radiology
ISSN
1071-3581
eISSN
1532-6551
D.O.I.
10.1007/s12350-016-0763-1
Publisher site
See Article on Publisher Site

Abstract

EDITORIAL Regadenoson-induced hyperemia for absolute myocardial blood flow quantitation by N- ammonia PET and detection of cardiac allograft vasculopathy a,b,c Rene R. Sevag Packard, MD, PhD, and Jamshid Maddahi, MD, FACC, a,b,d FASNC Division of Cardiology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA Ronald Reagan UCLA Medical Center, Los Angeles, CA Veterans Affairs West Los Angeles Medical Center, Los Angeles, CA Nuclear Medicine Clinic, Department of Molecular and Medical Pharmacology, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA Received Oct 4, 2016; accepted Oct 5, 2016 doi:10.1007/s12350-016-0763-1 available organs has led to the rise of ventricular assist See related article, pp. 1134–1144 devices implanted as destination therapy and to a very rigorous transplant recipient selection process. Fol- lowing OHT, transplant recipients are exposed to a In this issue of the Journal of Nuclear Cardiology, stringent surveillance program periodically screening for Miguel H. Pampolini and associates report on non-in- graft rejection, graft failure, infection, malignancy, and vasive quantitation of myocardial blood flow (MBF) by other complications. CAV, or transplant vasculopathy, regadenoson positron emission tomography (PET) accounts for *10% of deaths in OHT recipients starting imaging for

Journal

Journal of Nuclear CardiologySpringer Journals

Published: Jan 30, 2017

References

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