EDITORIAL Regadenoson-induced hyperemia for absolute myocardial blood ﬂow quantitation by N- ammonia PET and detection of cardiac allograft vasculopathy a,b,c Rene R. Sevag Packard, MD, PhD, and Jamshid Maddahi, MD, FACC, a,b,d FASNC Division of Cardiology, Department of Medicine, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA Ronald Reagan UCLA Medical Center, Los Angeles, CA Veterans Affairs West Los Angeles Medical Center, Los Angeles, CA Nuclear Medicine Clinic, Department of Molecular and Medical Pharmacology, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA Received Oct 4, 2016; accepted Oct 5, 2016 doi:10.1007/s12350-016-0763-1 available organs has led to the rise of ventricular assist See related article, pp. 1134–1144 devices implanted as destination therapy and to a very rigorous transplant recipient selection process. Fol- lowing OHT, transplant recipients are exposed to a In this issue of the Journal of Nuclear Cardiology, stringent surveillance program periodically screening for Miguel H. Pampolini and associates report on non-in- graft rejection, graft failure, infection, malignancy, and vasive quantitation of myocardial blood ﬂow (MBF) by other complications. CAV, or transplant vasculopathy, regadenoson positron emission tomography (PET) accounts for *10% of deaths in OHT recipients starting imaging for
Journal of Nuclear Cardiology – Springer Journals
Published: Jan 30, 2017
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