Persistent Genital Arousal Disorder: Current Conceptualizations and Etiologic Mechanisms

Persistent Genital Arousal Disorder: Current Conceptualizations and Etiologic Mechanisms Curr Sex Health Rep (2017) 9:177–182 FEMALE SEXUAL DYSFUNCTION AND DISORDERS (M CHIVERS AND C PUKALL, SECTION EDITORS) Persistent Genital Arousal Disorder: Current Conceptualizations and Etiologic Mechanisms 1 2 Barry R. Komisaruk & Irwin Goldstein Published online: 23 October 2017 Springer Science+Business Media, LLC 2017 Abstract symptoms in women and men and may be treatable. Chronic Purpose of review Persistent genital arousal disorder irritative radiculopathy could eventually affect nerve conduc- (PGAD), characterized in 2001, persists as a distressing mal- tion deleteriously and thereby attenuate genital afferent and/or ady that is not well appreciated, understood, or treated by efferent activity, leading to genital paresthesias, anorgasmia, healthcare providers. This review describes the characteristics or anejaculation. of PGAD, hypotheses regarding its etiology, therapies, and . . recent findings that provide evidence of PGAD as a genital Keywords Persistentgenitalarousaldisorder PGAD Tarlov . . . sensory neuropathy. cyst Cauda equina syndrome Genital nerve Anorgasmia Recent findings PGAD can result from (a) Tarlov cysts, which contain aberrant sensory nerve fibers and form on the genital sensory nerves where they abrade on the entrance to the sa- Introduction crum, and/or (b) herniated intervertebral disc-produced irrita- tion of the roots of those genital sensory nerves as Current Sexual Health Reports Springer Journals

Persistent Genital Arousal Disorder: Current Conceptualizations and Etiologic Mechanisms

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Copyright © 2017 by Springer Science+Business Media, LLC
Medicine & Public Health; Urology; Endocrinology
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