Oesophageal Squamous Dysplasia

Oesophageal Squamous Dysplasia Journal of Gastrointestinal Cancer (2018) 49:385–388 https://doi.org/10.1007/s12029-018-0122-3 EDITORIAL 1 1 2 Michael Auld & Havish Srinath & Eshwarshanker Jeyarajan Published online: 28 May 2018 Springer Science+Business Media, LLC, part of Springer Nature 2018 Epidemiology and Aetiology consumed regularly [3]. Several genetic differences in oe- sophageal SCC occur in Asian populations compared to Squamous cell carcinoma (SCC) of the oesophagus is the Caucasians which may contribute to the higher incidence most common neoplasia of the oesophagus worldwide [1]. identified [6]. Variances in enzyme functionality for alcohol In Australia, the incidence of oesophageal SCC is approxi- dehydrogenase, particularly amongst the Japanese popula- mately 1 in 100,000 with males having a higher predilection tion, may also contribute [7]. Numerous medical conditions [2]. The diagnosis is predominantly through endoscopic ex- predispose patients to the development of chronic oesoph- amination of the oesophagus. The overall incidence of oe- ageal inflammation, increasing their risk. Achalasia, sophageal SCC has been trending downward over the last Plummer-Vinson syndrome and scleroderma all increase several decades [3, 4]. Western countries, in particular, have the risk of oesophageal squamous dysplasia by oesophageal lower rates with some South African and Middle Eastern stasis [3]. An association between oesophageal carcinoma countries experiencing http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Gastrointestinal Cancer Springer Journals
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Publisher
Springer US
Copyright
Copyright © 2018 by Springer Science+Business Media, LLC, part of Springer Nature
Subject
Medicine & Public Health; Oncology; Gastroenterology; Radiotherapy; Cancer Research; Internal Medicine
ISSN
1941-6628
eISSN
1941-6636
D.O.I.
10.1007/s12029-018-0122-3
Publisher site
See Article on Publisher Site

Abstract

Journal of Gastrointestinal Cancer (2018) 49:385–388 https://doi.org/10.1007/s12029-018-0122-3 EDITORIAL 1 1 2 Michael Auld & Havish Srinath & Eshwarshanker Jeyarajan Published online: 28 May 2018 Springer Science+Business Media, LLC, part of Springer Nature 2018 Epidemiology and Aetiology consumed regularly [3]. Several genetic differences in oe- sophageal SCC occur in Asian populations compared to Squamous cell carcinoma (SCC) of the oesophagus is the Caucasians which may contribute to the higher incidence most common neoplasia of the oesophagus worldwide [1]. identified [6]. Variances in enzyme functionality for alcohol In Australia, the incidence of oesophageal SCC is approxi- dehydrogenase, particularly amongst the Japanese popula- mately 1 in 100,000 with males having a higher predilection tion, may also contribute [7]. Numerous medical conditions [2]. The diagnosis is predominantly through endoscopic ex- predispose patients to the development of chronic oesoph- amination of the oesophagus. The overall incidence of oe- ageal inflammation, increasing their risk. Achalasia, sophageal SCC has been trending downward over the last Plummer-Vinson syndrome and scleroderma all increase several decades [3, 4]. Western countries, in particular, have the risk of oesophageal squamous dysplasia by oesophageal lower rates with some South African and Middle Eastern stasis [3]. An association between oesophageal carcinoma countries experiencing

Journal

Journal of Gastrointestinal CancerSpringer Journals

Published: May 28, 2018

References

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