Mammalian Genome 9, 473–475 (1998). Incorporating Mouse Genome © Springer-Verlag New York Inc. 1998 Mapping of the human and murine X11-like genes (APBA2 and Apba2), the murine Fe65 gene (Apbb1), and the human Fe65-like gene (APBB2): genes encoding phosphotyrosine-binding domain proteins that interact with the Alzheimer’s disease amyloid precursor protein 1 2 1 2 2,3 Gonzalo Blanco, Nicholas G. Irving, Steve D.M. Brown, Christopher C.J. Miller, Declan M. McLoughlin MRC Mouse Genome Centre and MRC Mammalian Genetics Unit, Harwell, Didcot, Oxon OX11 ORD, UK Department of Neuroscience, Institute of Psychiatry, De Crespigny Park, Denmark Hill, London SE5 8AF, UK Section of Old Age Psychiatry, Institute of Psychiatry, De Crespigny Park, Denmark Hill, London SE5 8AF, UK Received: 16 December 1997 / Accepted: 20 February 1998 Abnormal processing of the membrane-spanning amyloid precur- (McLoughlin and Miller 1996) and overlapping sequences depos- sor protein (APP), resulting in the production of increased amounts ited in the databases (accession numbers R89683, R13010, of fibrillogenic b-amyloid peptide (Ab), is considered to be one of R18654, and T16098 for APBA2 and accession number the key metabolic events underlying Alzheimer’s disease (AD; HSU62325 for APBB2). For APBA2 the following primer pair: Selkoe 1994). The function of
Mammalian Genome – Springer Journals
Published: Jun 1, 1998
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