Involvement of the cerebral veins in MELAS syndrome?

Involvement of the cerebral veins in MELAS syndrome? Neuroradiology (2017) 59:833–834 DOI 10.1007/s00234-017-1883-2 LETTER TO THE EDITOR 1 2 Josef Finsterer & Sinda Zarrouk-Mahjoub Received: 5 July 2017 /Accepted: 5 July 2017 /Published online: 18 July 2017 Springer-Verlag GmbH Germany 2017 Dear Editor-in-Chief, MELAS [2]? Which were the clinical manifestations of the We read with interest the article by Whitehead et al. about a stroke? Which were the imaging findings of the Bstrokes^ retrospective study of 14 patients with MELAS, who had T2- during the acute phase? Were there typical abnormalities in- hyperintense cortical venous signals and unrelated T2-hyper- dicative of a cytotoxic edema on multimodal MRI or did the authors find a vasogenic edema? Did the white matter lesions intense, subcortical white matter lesions of unknown signifi- cance [1]. We have the following comments and concerns. or the venous signals disappear during follow-up? Did these The authors found abnormal cortical venous signals on T2/ lesions only occur during a stroke-like-episode? Which type FLAIR MRI and attributed it to thickening of the venous walls of treatment was applied for the Bstrokes^? Which is the and sluggish venous flow [1]. How to explain that abnormal pathomechanism underlying the venous abnormalities? venous wall has not been reported on autopsy http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Neuroradiology 0028-3940 Springer Journals

Involvement of the cerebral veins in MELAS syndrome?

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Publisher
Springer Berlin Heidelberg
Copyright
Copyright © 2017 by Springer-Verlag GmbH Germany
Subject
Medicine & Public Health; Neuroradiology; Imaging / Radiology; Neurology; Neurosurgery; Neurosciences
ISSN
0028-3940
eISSN
1432-1920
D.O.I.
10.1007/s00234-017-1883-2
Publisher site
See Article on Publisher Site

Abstract

Neuroradiology (2017) 59:833–834 DOI 10.1007/s00234-017-1883-2 LETTER TO THE EDITOR 1 2 Josef Finsterer & Sinda Zarrouk-Mahjoub Received: 5 July 2017 /Accepted: 5 July 2017 /Published online: 18 July 2017 Springer-Verlag GmbH Germany 2017 Dear Editor-in-Chief, MELAS [2]? Which were the clinical manifestations of the We read with interest the article by Whitehead et al. about a stroke? Which were the imaging findings of the Bstrokes^ retrospective study of 14 patients with MELAS, who had T2- during the acute phase? Were there typical abnormalities in- hyperintense cortical venous signals and unrelated T2-hyper- dicative of a cytotoxic edema on multimodal MRI or did the authors find a vasogenic edema? Did the white matter lesions intense, subcortical white matter lesions of unknown signifi- cance [1]. We have the following comments and concerns. or the venous signals disappear during follow-up? Did these The authors found abnormal cortical venous signals on T2/ lesions only occur during a stroke-like-episode? Which type FLAIR MRI and attributed it to thickening of the venous walls of treatment was applied for the Bstrokes^? Which is the and sluggish venous flow [1]. How to explain that abnormal pathomechanism underlying the venous abnormalities? venous wall has not been reported on autopsy

Journal

Neuroradiology 0028-3940Springer Journals

Published: Jul 18, 2017

References

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