Highlight report: N-acetyltransferase 2 and urinary bladder cancer risk

Highlight report: N-acetyltransferase 2 and urinary bladder cancer risk Arch Toxicol (2017) 91:3205–3206 DOI 10.1007/s00204-017-2001-2 EDITORIAL Highlight report: N‑acetyltransferase 2 and urinary bladder cancer risk Meinolf Blaszkewicz Received: 24 May 2017 / Accepted: 29 May 2017 / Published online: 2 June 2017 © Springer-Verlag Berlin Heidelberg 2017 In recent years, genome-wide association studies have bladder cancer is currently hampered by the lack of large identified and validated a set of genetic variants that are cohorts with available DNA and a carefully established associated with urinary bladder cancer risk (e.g., Figueroa follow-up. Nevertheless, one of the most important steps in et al. 2016; Rafnar et al. 2014; Selinski 2014a, b; Selin- future will be to analyze also all other risk variants iden- ski et al. 2012, 2013; Kiemeney et al. 2010; review: Golka tified in genome-wide association studies with respect to et al. 2011). After identification of the most relevant indi - their prognostic relevance after surgery of bladder cancer. vidual variants, it became possible to identify SNP interac- tions (Schwender et al. 2015) and to differentiate the contri- bution of genetic predisposition and occupational exposure References to urinary bladder cancer risk (Selinski et al. 2016; Krech Figueroa JD, Middlebrooks CD, Banday AR, Silverman DT, et al. 2013, 2016). http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Archives of Toxicology Springer Journals

Highlight report: N-acetyltransferase 2 and urinary bladder cancer risk

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Publisher
Springer Berlin Heidelberg
Copyright
Copyright © 2017 by Springer-Verlag Berlin Heidelberg
Subject
Biomedicine; Pharmacology/Toxicology; Occupational Medicine/Industrial Medicine; Environmental Health; Biomedicine, general
ISSN
0340-5761
eISSN
1432-0738
D.O.I.
10.1007/s00204-017-2001-2
Publisher site
See Article on Publisher Site

Abstract

Arch Toxicol (2017) 91:3205–3206 DOI 10.1007/s00204-017-2001-2 EDITORIAL Highlight report: N‑acetyltransferase 2 and urinary bladder cancer risk Meinolf Blaszkewicz Received: 24 May 2017 / Accepted: 29 May 2017 / Published online: 2 June 2017 © Springer-Verlag Berlin Heidelberg 2017 In recent years, genome-wide association studies have bladder cancer is currently hampered by the lack of large identified and validated a set of genetic variants that are cohorts with available DNA and a carefully established associated with urinary bladder cancer risk (e.g., Figueroa follow-up. Nevertheless, one of the most important steps in et al. 2016; Rafnar et al. 2014; Selinski 2014a, b; Selin- future will be to analyze also all other risk variants iden- ski et al. 2012, 2013; Kiemeney et al. 2010; review: Golka tified in genome-wide association studies with respect to et al. 2011). After identification of the most relevant indi - their prognostic relevance after surgery of bladder cancer. vidual variants, it became possible to identify SNP interac- tions (Schwender et al. 2015) and to differentiate the contri- bution of genetic predisposition and occupational exposure References to urinary bladder cancer risk (Selinski et al. 2016; Krech Figueroa JD, Middlebrooks CD, Banday AR, Silverman DT, et al. 2013, 2016).

Journal

Archives of ToxicologySpringer Journals

Published: Jun 2, 2017

References

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