Short Communications Incorporating Mouse Genome Mammalian Genome 10, 1017–1019 (1999). © Springer-Verlag New York Inc. 1999 Genetic mapping of six mouse peroxiredoxin genes and fourteen peroxiredoxin related sequences 1, 2 2, 2, 1 2 Myung S. Lyu, * Sue Goo Rhee, Ho Zoon Chae, ** Tae Hoon Lee, † M. Charlene Adamson, Sang Won Kang, 1, 1 1 Dong-Yan Jin, ‡ Kuan-Teh Jeang, Christine A. Kozak Laboratory of Molecular Microbiology, National Institute of Allergy and Infectious Diseases, NIH, Building 4, Room 329, 4 Center Drive MSC 0460, Bethesda, MD 20892-0460, USA Laboratory of Cell Signaling, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Maryland 20892, USA Received: 5 March 1999 / Accepted: 8 June 1999 Organisms living in aerobic environments require mechanisms that gous recombination (Chae et al. 1993). The mutant and wild-type prevent or limit cellular damage caused by reactive oxygen species strains grew at equal rates under anaerobic conditions. However, (O ,H O , and HO) that arise from the incomplete reduction of under aerobic conditions, especially under oxidative stress, the 2 2 2 oxygen during respiration. Alternatively, damage can result from growth rate of mutant yeast was significantly lower than that of exposure
Mammalian Genome – Springer Journals
Published: Oct 1, 1999
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