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13/Jammalian Short Communications Genome Incorporating Moose Groom, Mammalian Genome 9, 74-75 (1998). Springer-Verlag New York Inc. 1998 Cloning, expression and genetic mapping of the mouse SH3 domain protein, SH3D2B Ted Torrey,' Wankee Kim,' Herbert C. Morse III,' Christine A. Kozak 2 'Laboratory of Immunopathology, National Institute of Allergy and Infectious Diseases, National Institute of Health, Bethesda, Maryland 20892, USA 2Laboratory of Molecular Microbiology, National Institute of Allergy and Infectious Diseases, Building 4, Room 329, National Institutes of Health, Bethesda, Maryland 20892-0460, USA Received: 24 July 1997 / Accepted: 19 September 1997 The LP-BM5 mixture of murine leukemia viruses causes an im- the 2.7-kb single transcript found ubiquitously in human tissues munodeficiency syndrome in mice termed MAIDS (reviewed in with SH3GL1 as probe (Giachino et al. 1997). Morse et al. 1992). The etiological agent in the mixture is a rep- Sh3d2b was genetically mapped by analysis of two sets of lication-defective virus (BM5def) that encodes only a single pro- genetic crosses: (NFS/N or C581J x M. m. musculus) x M. m. tein. This protein is a variant of the normal viral Gag precursor that musculus (Kozak et al. 1990), (NFS/N x M. spretus) x M. spretus is cleaved into the
Mammalian Genome – Springer Journals
Published: Jan 1, 1998
Keywords: Acute Myeloid Leukemia; Viral Integration Site; Ecotropic Virus; Human Gene Family; Leukemia Virus Receptor
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