Autoantibodies binding to stathmin-4: new marker for polyneuropathy in primary Sjögren’s syndrome

Autoantibodies binding to stathmin-4: new marker for polyneuropathy in primary Sjögren’s syndrome Immunol Res (2017) 65:1099–1102 https://doi.org/10.1007/s12026-017-8970-7 BRIEF REPORT Autoantibodies binding to stathmin-4: new marker for polyneuropathy in primary Sjögren’s syndrome 1 1 2 1 1 Sabrina Duda & Torsten Witte & Martin Stangel & Jan Adams & Reinhold E. Schmidt & Niklas T. Baerlecken Published online: 14 November 2017 Springer Science+Business Media, LLC, part of Springer Nature 2017 Introduction Methods Primary Sjögren’s syndrome (pSS) is an autoimmune disease Patients, materials, and methods affecting exocrine glands [1] and may be associated with dif- ferent extraglandular manifestations such as arthritis, myosi- We collected sera of patients with pSS (n =72), with second- tis, vasculitis, and peripheral polyneuropathy (PNP) or central ary Sjögren’ssyndrome(sSS, n =20), SLE (n =36), rheuma- nervous system affection [2]. toid arthritis (RA, n = 62), granulomatosis with polyangiitis A precise diagnosis of associated neurological manifesta- (GPA, n = 21), undifferentiated connective tissue disease tions is still a challenging endeavor. 8.5–67% of patients with (UCTD, n = 27), multiple sclerosis (MS, n = 37), metabolic pSS complain about neurological symptoms. In approximate- and toxic PNP (n = 47), multiple myeloma (n = 44), and of ly 9% of the patients, a polyneuropathy is diagnosed [3–5]. blood donors (BD, n = http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Immunologic Research Springer Journals

Autoantibodies binding to stathmin-4: new marker for polyneuropathy in primary Sjögren’s syndrome

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Publisher
Springer Journals
Copyright
Copyright © 2017 by Springer Science+Business Media, LLC, part of Springer Nature
Subject
Medicine & Public Health; Allergology; Immunology; Medicine/Public Health, general; Internal Medicine
ISSN
0257-277X
eISSN
1559-0755
D.O.I.
10.1007/s12026-017-8970-7
Publisher site
See Article on Publisher Site

Abstract

Immunol Res (2017) 65:1099–1102 https://doi.org/10.1007/s12026-017-8970-7 BRIEF REPORT Autoantibodies binding to stathmin-4: new marker for polyneuropathy in primary Sjögren’s syndrome 1 1 2 1 1 Sabrina Duda & Torsten Witte & Martin Stangel & Jan Adams & Reinhold E. Schmidt & Niklas T. Baerlecken Published online: 14 November 2017 Springer Science+Business Media, LLC, part of Springer Nature 2017 Introduction Methods Primary Sjögren’s syndrome (pSS) is an autoimmune disease Patients, materials, and methods affecting exocrine glands [1] and may be associated with dif- ferent extraglandular manifestations such as arthritis, myosi- We collected sera of patients with pSS (n =72), with second- tis, vasculitis, and peripheral polyneuropathy (PNP) or central ary Sjögren’ssyndrome(sSS, n =20), SLE (n =36), rheuma- nervous system affection [2]. toid arthritis (RA, n = 62), granulomatosis with polyangiitis A precise diagnosis of associated neurological manifesta- (GPA, n = 21), undifferentiated connective tissue disease tions is still a challenging endeavor. 8.5–67% of patients with (UCTD, n = 27), multiple sclerosis (MS, n = 37), metabolic pSS complain about neurological symptoms. In approximate- and toxic PNP (n = 47), multiple myeloma (n = 44), and of ly 9% of the patients, a polyneuropathy is diagnosed [3–5]. blood donors (BD, n =

Journal

Immunologic ResearchSpringer Journals

Published: Nov 14, 2017

References

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