Immunol Res (2017) 65:1099–1102 https://doi.org/10.1007/s12026-017-8970-7 BRIEF REPORT Autoantibodies binding to stathmin-4: new marker for polyneuropathy in primary Sjögren’s syndrome 1 1 2 1 1 Sabrina Duda & Torsten Witte & Martin Stangel & Jan Adams & Reinhold E. Schmidt & Niklas T. Baerlecken Published online: 14 November 2017 Springer Science+Business Media, LLC, part of Springer Nature 2017 Introduction Methods Primary Sjögren’s syndrome (pSS) is an autoimmune disease Patients, materials, and methods affecting exocrine glands  and may be associated with dif- ferent extraglandular manifestations such as arthritis, myosi- We collected sera of patients with pSS (n =72), with second- tis, vasculitis, and peripheral polyneuropathy (PNP) or central ary Sjögren’ssyndrome(sSS, n =20), SLE (n =36), rheuma- nervous system affection . toid arthritis (RA, n = 62), granulomatosis with polyangiitis A precise diagnosis of associated neurological manifesta- (GPA, n = 21), undifferentiated connective tissue disease tions is still a challenging endeavor. 8.5–67% of patients with (UCTD, n = 27), multiple sclerosis (MS, n = 37), metabolic pSS complain about neurological symptoms. In approximate- and toxic PNP (n = 47), multiple myeloma (n = 44), and of ly 9% of the patients, a polyneuropathy is diagnosed [3–5]. blood donors (BD, n =
Immunologic Research – Springer Journals
Published: Nov 14, 2017
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