Plant Mol Biol (2016) 92:613–628 DOI 10.1007/s1103-016-0537-91 ed levels of Alter AtHSCB disrupts iron translocation from roots to shoots 1,2 1 1 1 Laura Leaden · María A. Pagani · Manuel Balparda · María .V Busi · Diego F. Gomez-Casati Received: 6 November 2015 / Accepted: 29 August 2016 / Published online: 21 September 2016 © Springer Science +Business Media Dordrecht 2016 Abstract another which need to be identified. Here, we character - Key message Plants overessing expr AtHSCB and hscb ized Arabidopsis thaliana plants overexpressing AtHSCB, knockdown mutants showed altered iron homeosta - encoding a mitochondrial cochaperone involved in [Fe–S] sis. The overession of expr AtHSCB led to activation of cluster biosynthesis, and hscb knockdown mutants, which the iron uptake system and iron accumulation in roots exhibit altered shoot/root Fe partitioning. Overexpression of without concomitant transport to shoots, resulting in AtHSCB induced an increase in root iron uptake and con- educed irr on content in the aerial parts of plants. By tent along with iron deficiency in shoots. Conversely, hscb contrast, hscb knockdown mutants presented the oppo - knockdown mutants exhibited increased iron accumulation site phenotype, with iron accumulation in shoots despite in shoots and reduced iron uptake
Plant Molecular Biology – Springer Journals
Published: Sep 21, 2016
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