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Eur J Pediatr (2004) 163: 341–342 DOI 10.1007/s00431-004-1433-x SHORT REPORT Toru Watanabe Ulcerative colitis that developed 8 years after human parvovirus B19 encephalopathy Received: 30 October 2003 / Accepted: 19 February 2004 / Published online: 23 March 2004 Springer-Verlag 2004 fluid studies demonstrated lymphocyte dominant, mild A 13-year-old girl suffered from ulcerative colitis, 8 years after human parvovirus B19 encephalopathy. pleocytosis (9 cells/ll), an increase in protein concen- tration (46 mg/dl), and negative B19-DNA results. Ulcerative colitis is a chronic inflammatory disease of B19-associated encephalopathy was diagnosed [7]. the rectum and colon [3] and its incidence has recently increased in children [4]. Several factors are postulated Subsequently, secondary localisation-related epilepsy and mental retardation developed and valproic acid to contribute to the development of ulcerative colitis and primidone were prescribed, but could not control including environmental, microbial, genetic and immune factors [3]. her seizures completely. Eight years later, she was referred to our hospital Since we first described human parvovirus B19 (B19)- because she had been suffering from abdominal pain, associated encephalopathy in 1994 [7], B19-associated encephalitis/encephalopathy has occasionally been bloody diarrhoea, low-grade fever and body weight loss during the preceding 3 months. The patient was anae- reported [1, 2,
European Journal of Pediatrics – Springer Journals
Published: Mar 23, 2004
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