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Erratum to: dysregulation of matricellular proteins is an early signature of pathology in laminindeficient muscular dystrophy

Erratum to: dysregulation of matricellular proteins is an early signature of pathology in... Mehuron et al. Skeletal Muscle 2014, 4:17 http://www.skeletalmusclejournal.com/content/4/1/17 CORRECTION Open Access Correction: dysregulation of matricellular proteins is an early signature of pathology in laminin- deficient muscular dystrophy Thomas Mehuron , Ajay Kumar, Lina Duarte, Jenny Yamauchi, Anthony Accorsi and Mahasweta Girgenrath Correction After publication of this work [1], we noted that while we were initially going to include muscle function tests in this study, as our manuscript evolved and we decided to include earlier time points for characterization, we decided to remove it because pups younger than 2 weeks cannot be measured. While we removed this from the results, we missed retracting them from the methods section of the abstract. Therefore, the methods section of the abstract should instead read: Methods We sought out to examine the dysregulation of various pathways throughout early development (postnatal weeks 1–4) in the DyW mouse, the most commonly used mouse model of laminin-deficient muscular dystrophy. Gene (qRT-PCR) and protein levels (western blot, ELISA) as well as histology (H&E, picrosirius red staining) and immuno- histochemistry (fibronectin, TUNEL assay) were used to assess dysregulation of matricellular proteins. Received: 18 September 2014 Accepted: 18 September 2014 Published: 2 October 2014 References 1. Mehuron T, Kumar A, Duarte L, Yamauchi J, Accorsi A, Girgenrath M: Dysregulation of matricellular proteins is an early signature of pathology in laminin-deficient muscular dystrophy. Skeletal Muscle 2014, 4:14. doi:10.1186/2044-5040-4-17 Submit your next manuscript to BioMed Central Cite this article as: Mehuron et al.: Correction: dysregulation of matricellular and take full advantage of: proteins is an early signature of pathology in laminin-deficient muscular dystrophy. Skeletal Muscle 2014 4:17. • Convenient online submission • Thorough peer review • No space constraints or color figure charges • Immediate publication on acceptance • Inclusion in PubMed, CAS, Scopus and Google Scholar • Research which is freely available for redistribution * Correspondence: tmehuron@bu.edu Submit your manuscript at Department of Health Sciences, Boston University, 635 Commonwealth www.biomedcentral.com/submit Avenue, Boston, MA 02215, USA © 2014 Mehuron et al.; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Skeletal Muscle Springer Journals

Erratum to: dysregulation of matricellular proteins is an early signature of pathology in laminindeficient muscular dystrophy

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Publisher
Springer Journals
Copyright
Copyright © 2014 by Mehuron et al.; licensee BioMed Central Ltd.
Subject
Life Sciences; Cell Biology; Developmental Biology; Biochemistry, general; Systems Biology; Biotechnology
eISSN
2044-5040
DOI
10.1186/2044-5040-4-17
Publisher site
See Article on Publisher Site

Abstract

Mehuron et al. Skeletal Muscle 2014, 4:17 http://www.skeletalmusclejournal.com/content/4/1/17 CORRECTION Open Access Correction: dysregulation of matricellular proteins is an early signature of pathology in laminin- deficient muscular dystrophy Thomas Mehuron , Ajay Kumar, Lina Duarte, Jenny Yamauchi, Anthony Accorsi and Mahasweta Girgenrath Correction After publication of this work [1], we noted that while we were initially going to include muscle function tests in this study, as our manuscript evolved and we decided to include earlier time points for characterization, we decided to remove it because pups younger than 2 weeks cannot be measured. While we removed this from the results, we missed retracting them from the methods section of the abstract. Therefore, the methods section of the abstract should instead read: Methods We sought out to examine the dysregulation of various pathways throughout early development (postnatal weeks 1–4) in the DyW mouse, the most commonly used mouse model of laminin-deficient muscular dystrophy. Gene (qRT-PCR) and protein levels (western blot, ELISA) as well as histology (H&E, picrosirius red staining) and immuno- histochemistry (fibronectin, TUNEL assay) were used to assess dysregulation of matricellular proteins. Received: 18 September 2014 Accepted: 18 September 2014 Published: 2 October 2014 References 1. Mehuron T, Kumar A, Duarte L, Yamauchi J, Accorsi A, Girgenrath M: Dysregulation of matricellular proteins is an early signature of pathology in laminin-deficient muscular dystrophy. Skeletal Muscle 2014, 4:14. doi:10.1186/2044-5040-4-17 Submit your next manuscript to BioMed Central Cite this article as: Mehuron et al.: Correction: dysregulation of matricellular and take full advantage of: proteins is an early signature of pathology in laminin-deficient muscular dystrophy. Skeletal Muscle 2014 4:17. • Convenient online submission • Thorough peer review • No space constraints or color figure charges • Immediate publication on acceptance • Inclusion in PubMed, CAS, Scopus and Google Scholar • Research which is freely available for redistribution * Correspondence: tmehuron@bu.edu Submit your manuscript at Department of Health Sciences, Boston University, 635 Commonwealth www.biomedcentral.com/submit Avenue, Boston, MA 02215, USA © 2014 Mehuron et al.; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

Journal

Skeletal MuscleSpringer Journals

Published: Oct 2, 2014

References