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Correction to: ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 transcription in nasopharyngeal carcinoma

Correction to: ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity... Peng et al. J Exp Clin Cancer Res (2022) 41:30 https://doi.org/10.1186/s13046-021-02238-5 Open Access CORREC TION Correction to: ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 transcription in nasopharyngeal carcinoma 1† 2† 2† 1 1 2 2 Hao Peng , Jian Zhang , Pan‑Pan Zhang , Lei Chen , Ling‑Long Tang , XiaoJing ‑ Yang , Qing‑Mei He , 2 1 2 2* 1* Xin Wen , Ying Sun , Na Liu , Ying‑Qin Li and Jun Ma Correction to: J Exp Clin Cancer Res 38, 11 (2019) Supplementary Information https://doi.org/10.1186/s13046-018-0997-7 The online version contains supplementary material available at https:// doi. org/ 10. 1186/ s13046‑ 021‑ 02238‑5. Following publication of the original article [1], the Additional file 2: Fig. S1. ARNTL methylation levels in the GSE52068 and authors identified some minor errors in Supplemental GSE62366 nasopharyngeal carcinoma datasets. Figs. 1 and 4, specifically: Additional file 5: Fig. S4. Overexpression of ARNTL had no impact on nasopharyngeal carcinoma cells invasion and migration. (A) Images of Transwell invasion (left) and migration (right) assay with ARNTL‑ overex‑ • Fig. S1b: incorrect sample size listed; correct sample pression or Vector‑ overexpression SUNE1 and HONE1 cells. (B) Images of size is 25 wound healing assay with ARNTL‑ overexpression or Vector‑ overexpression • Fig. S4b: incorrect image used for SUNE1-ARNTL SUNE1 and HONE1 cells. (24 h); correct image is now used The authors provided the journal with their original data. The corrected figures are given here. The correc - tions do not have any effect on the final conclusions of the paper. The original article has been corrected. Reference 1. Peng H, Zhang J, Zhang PP, et al. ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 tran‑ scription in nasopharyngeal carcinoma. J Exp Clin Cancer Res. 2019;38:11. The original article can be found online at https:// doi. org/ 10. 1186/ s13046‑ https:// doi. org/ 10. 1186/ s13046‑ 018‑ 0997‑7. 018‑ 0997‑7. *Correspondence: liyingq@sysucc.org.cn; majun2@mail.sysu.edu.cn Hao Peng, Jian Zhang and Pan‑Pan Zhang contributed equally to this work. Department of Radiation Oncology, State Key Laboratory of Oncology in Southern China, Collaborative Innovation Center for Cancer Medicine, Guangdong Key Laboratory of Nasopharyngeal Carcinoma Diagnosis and Therapy, Sun Yat‑sen University Cancer Center, Guangdong 510060, People’s Republic of China Department of Experimental Research, State Key Laboratory of Oncology in Southern China, Collaborative Innovation Center for Cancer Medicine, Guangdong Key Laboratory of Nasopharyngeal Carcinoma Diagnosis and Therapy, Sun Yat‑sen University Cancer Center, Guangdong 510060, People’s Republic of China © The Author(s) 2022. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http:// creat iveco mmons. org/ licen ses/ by/4. 0/. The Creative Commons Public Domain Dedication waiver (http:// creat iveco mmons. org/ publi cdoma in/ zero/1. 0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Experimental & Clinical Cancer Research Springer Journals

Correction to: ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 transcription in nasopharyngeal carcinoma

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Publisher
Springer Journals
Copyright
Copyright © The Author(s) 2022
eISSN
1756-9966
DOI
10.1186/s13046-021-02238-5
Publisher site
See Article on Publisher Site

Abstract

Peng et al. J Exp Clin Cancer Res (2022) 41:30 https://doi.org/10.1186/s13046-021-02238-5 Open Access CORREC TION Correction to: ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 transcription in nasopharyngeal carcinoma 1† 2† 2† 1 1 2 2 Hao Peng , Jian Zhang , Pan‑Pan Zhang , Lei Chen , Ling‑Long Tang , XiaoJing ‑ Yang , Qing‑Mei He , 2 1 2 2* 1* Xin Wen , Ying Sun , Na Liu , Ying‑Qin Li and Jun Ma Correction to: J Exp Clin Cancer Res 38, 11 (2019) Supplementary Information https://doi.org/10.1186/s13046-018-0997-7 The online version contains supplementary material available at https:// doi. org/ 10. 1186/ s13046‑ 021‑ 02238‑5. Following publication of the original article [1], the Additional file 2: Fig. S1. ARNTL methylation levels in the GSE52068 and authors identified some minor errors in Supplemental GSE62366 nasopharyngeal carcinoma datasets. Figs. 1 and 4, specifically: Additional file 5: Fig. S4. Overexpression of ARNTL had no impact on nasopharyngeal carcinoma cells invasion and migration. (A) Images of Transwell invasion (left) and migration (right) assay with ARNTL‑ overex‑ • Fig. S1b: incorrect sample size listed; correct sample pression or Vector‑ overexpression SUNE1 and HONE1 cells. (B) Images of size is 25 wound healing assay with ARNTL‑ overexpression or Vector‑ overexpression • Fig. S4b: incorrect image used for SUNE1-ARNTL SUNE1 and HONE1 cells. (24 h); correct image is now used The authors provided the journal with their original data. The corrected figures are given here. The correc - tions do not have any effect on the final conclusions of the paper. The original article has been corrected. Reference 1. Peng H, Zhang J, Zhang PP, et al. ARNTL hypermethylation promotes tumorigenesis and inhibits cisplatin sensitivity by activating CDK5 tran‑ scription in nasopharyngeal carcinoma. J Exp Clin Cancer Res. 2019;38:11. The original article can be found online at https:// doi. org/ 10. 1186/ s13046‑ https:// doi. org/ 10. 1186/ s13046‑ 018‑ 0997‑7. 018‑ 0997‑7. *Correspondence: liyingq@sysucc.org.cn; majun2@mail.sysu.edu.cn Hao Peng, Jian Zhang and Pan‑Pan Zhang contributed equally to this work. Department of Radiation Oncology, State Key Laboratory of Oncology in Southern China, Collaborative Innovation Center for Cancer Medicine, Guangdong Key Laboratory of Nasopharyngeal Carcinoma Diagnosis and Therapy, Sun Yat‑sen University Cancer Center, Guangdong 510060, People’s Republic of China Department of Experimental Research, State Key Laboratory of Oncology in Southern China, Collaborative Innovation Center for Cancer Medicine, Guangdong Key Laboratory of Nasopharyngeal Carcinoma Diagnosis and Therapy, Sun Yat‑sen University Cancer Center, Guangdong 510060, People’s Republic of China © The Author(s) 2022. Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http:// creat iveco mmons. org/ licen ses/ by/4. 0/. The Creative Commons Public Domain Dedication waiver (http:// creat iveco mmons. org/ publi cdoma in/ zero/1. 0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

Journal

Journal of Experimental & Clinical Cancer ResearchSpringer Journals

Published: Jan 21, 2022

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