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A commentary on the Third International Alzheimer's Disease Conference in Abano Termi, Italy

Sounding board A commentary on the Third International Alzheimer's Disease Conference in Abano Termi, Italy F. Marott Sinex, PhD I recently returned from the third International Alzheimer's Disease Conference in Abano Termi, Italy, held from July 12-17th, 1992. This was a large conference where one cannot hear everything, and what one hears may be biased by what one wants to hear. I was impressed by what has been accomplished since the Toronto Con- ference two years ago. There is a much clearer scenario for the development of AD. Information discerned from the con- ference indicates that the disease probably begins in the trans entorhinal region and spreads from there through the limbic system and finally to the association cortex. The first stages of the disease are silent and are charac- terized by tangles and neuronal loss, but not plaque formation. As the limbic system, the hippocampus, the amyg- dala, and the basal forebrain become more involved with neuronal loss and tangles, plaques may appear. The symptoms that are so distressing to families begin - depression, anxiety, hallucinations, wandering, and screaming are potentially treatable and Fi MarottSinex, PhD, DepartmentofBiochem- istry, Boston University School of Medicine, Boston, Massachusetts, and Board http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png American Journal of Alzheimer's Disease and Other Dementias SAGE

A commentary on the Third International Alzheimer's Disease Conference in Abano Termi, Italy

Abstract

Sounding board A commentary on the Third International Alzheimer's Disease Conference in Abano Termi, Italy F. Marott Sinex, PhD I recently returned from the third International Alzheimer's Disease Conference in Abano Termi, Italy, held from July 12-17th, 1992. This was a large conference where one cannot hear everything, and what one hears may be biased by what one wants to hear. I was impressed by what has been accomplished since the Toronto Con- ference two years ago. There is a much clearer scenario for the development of AD. Information discerned from the con- ference indicates that the disease probably begins in the trans entorhinal region and spreads from there through the limbic system and finally to the association cortex. The first stages of the disease are silent and are charac- terized by tangles and neuronal loss, but not plaque formation. As the limbic system, the hippocampus, the amyg- dala, and the basal forebrain become more involved with neuronal loss and tangles, plaques may appear. The symptoms that are so distressing to families begin - depression, anxiety, hallucinations, wandering, and screaming are potentially treatable and Fi MarottSinex, PhD, DepartmentofBiochem- istry, Boston University School of Medicine, Boston, Massachusetts, and Board
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