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METTL14 Promotes Proliferation, Migration, and Invasion in Endometriotic Stromal Cell Growth by Activating the ZEB1/MEK/ERK Pathway.

METTL14 Promotes Proliferation, Migration, and Invasion in Endometriotic Stromal Cell Growth by... The aim of this study was to explore the mechanism of methyltransferase-like 14 (METTL14) on human endometriotic stromal cell (ESC; HEM15A) proliferation, migration, and invasion to provide novel therapy for endometriosis (EMs). http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Gynecologic and Obstetric Investigation Pubmed

METTL14 Promotes Proliferation, Migration, and Invasion in Endometriotic Stromal Cell Growth by Activating the ZEB1/MEK/ERK Pathway.

Gynecologic and Obstetric Investigation : 13113 – Aug 21, 2024

METTL14 Promotes Proliferation, Migration, and Invasion in Endometriotic Stromal Cell Growth by Activating the ZEB1/MEK/ERK Pathway.


Abstract

The aim of this study was to explore the mechanism of methyltransferase-like 14 (METTL14) on human endometriotic stromal cell (ESC; HEM15A) proliferation, migration, and invasion to provide novel therapy for endometriosis (EMs).

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Copyright
© 2024 S. Karger AG, Basel.
eISSN
1423-002X
DOI
10.1159/000539656
pmid
38934184
Publisher site
See Article on Publisher Site

Abstract

The aim of this study was to explore the mechanism of methyltransferase-like 14 (METTL14) on human endometriotic stromal cell (ESC; HEM15A) proliferation, migration, and invasion to provide novel therapy for endometriosis (EMs).

Journal

Gynecologic and Obstetric InvestigationPubmed

Published: Aug 21, 2024

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