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Live discussion: Amyloid-ß degradation: The forgotten half of Alzheimer's disease

Journal of Alzheimer’s Disease 5 (2003) 491–497 IOS Press Discussion 1 Live Chat held 12 September 2002 with Wes Farris and Malcolm Leissring, Brigham and Women’s Hospital and Harvard Medical School. Posted 11 October 2002. Participants: Chastity Whitaker (Ohio State University), Chris Eckman (Mayo Clinic, Jacksonville), Claudia Almeida (Cornell University), Douglas Feinstein (University of Illinois, Chicago), Craig Atwood (Case Western Reserve University), Elizabeth Eckman (Mayo Clinic, Jacksonville), Keith Crutcher (University of Cincinnati), Lou Hersh (University of Kentucky), Malcolm Leissring (Harvard Medical School), Matthew LaVoie (Harvard Medical School), Nilufer Ertekin-Taner (Mayo Clinic, Jacksonville), Paul Shapiro (Elan Corporation), Reisuke Takahashi (Cornell University), Rina Yamin (Boston University School of Medicine), Stefan Mansourian (Harvard Medical ´ School), Steve Estus (University of Kentucky), Sylvain Lesn e (University of Caen, France), Tony Turner (Karolinski Institute, Stockholm, Sweden), Wes Farris (Harvard Medical School). Moderator: Gabrielle Strobel Malcolm Leissring and Wes Farris: We will start by discussing what genetic evidence there might be for the involvement of decreased amyloid-β (Aβ) degradation as a cause of Alzheimer’s disease (AD). Chris Eckman: Nilufer, this question may be best for you. Nilufer Ertekin-Taner: The main lines of genetic evidence, in my opinion, come from the mouse/rat knockout/mutant studies showing http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Journal of Alzheimer's Disease IOS Press

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