•UVB irradiation and activation of Toll-like receptor 3 by Poly I:C synergistically induce cell death in HaCaT cell.•Cell death induced by UVB-Poly I:C co-treatment is caspase-dependent apoptosis.•UVB-Poly I:C co-treatment enhances NF-κB activation through TLR3.•NF-κB pathway is responsible for apoptosis induced by UVB-Poly I:C.
Molecular Immunology – Elsevier
Published: Jul 1, 2018
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