C33-A cells transfected with E6*I or E6*II the short forms of HPV-16 E6, displayed opposite effects on cisplatin-induced apoptosis

C33-A cells transfected with E6*I or E6*II the short forms of HPV-16 E6, displayed opposite... •HPV-16 E6/E7 oncogenes are altenatively spliced in 3 products, E6*I, E6*II & E6^E7.•The role of HPV-16 E6 to produce apoptosis resistance in infected cells, is known.•The role of the E6 short forms on apoptosis resistance is unclear.•C·33-A cells transfected with the E6 short forms shown differential apoptosis.•E6*I and E6*II displayed opposite effects on cisplatin-induced apoptosis. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Virus Research Elsevier

C33-A cells transfected with E6*I or E6*II the short forms of HPV-16 E6, displayed opposite effects on cisplatin-induced apoptosis

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Publisher
Elsevier
Copyright
Copyright © 2018 Elsevier B.V.
ISSN
0168-1702
D.O.I.
10.1016/j.virusres.2018.02.009
Publisher site
See Article on Publisher Site

Abstract

•HPV-16 E6/E7 oncogenes are altenatively spliced in 3 products, E6*I, E6*II & E6^E7.•The role of HPV-16 E6 to produce apoptosis resistance in infected cells, is known.•The role of the E6 short forms on apoptosis resistance is unclear.•C·33-A cells transfected with the E6 short forms shown differential apoptosis.•E6*I and E6*II displayed opposite effects on cisplatin-induced apoptosis.

Journal

Virus ResearchElsevier

Published: Mar 2, 2018

References

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