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(1969)
National Cancer Institute monograph no. 30), ed. by S
R. Olson, R. Nordquist (1966)
Ultramicroscopic localization of acid phosphatase in human epidermis.The Journal of investigative dermatology, 46 4
M. Black, J. Mcquillin, P. Gardner (1972)
A rapid diagnostic aid for herpesvirus hominis infections in dermatology.Dermatologica, 144 1
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Rapid Diagnosis of Herpesvirus Hominis Infections in Superficial Lesions by Immunofluorescent Antibody TechniquesBritish Medical Journal, 4
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Cell loss as a factor in the growth rate of human tumours.European journal of cancer, 3 4
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Inflammatory changes in tumour vessels after systemic 5-hydroxytryptamine, bradykinin, kallikrein, or lysolecithin.British Journal of Cancer, 20
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A histochemical study of hypertrophy and ischaemic injury of rat liver with special reference to changes in lysosomes.The Journal of pathology and bacteriology, 90 2
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carcinomas that contain numerous mitotic figures
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Kinetics of cellular proliferation and cell loss in human carcinomas. A discussion of methods available for in vivo studies.European journal of cancer, 3 4
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Studies on tumor cell population kinetics
G. Weinstein, P. Frost (1970)
Cell proliferation in human basal cell carcinoma.Cancer research, 30 3
B. Vestergaard, H. Thybo (1974)
Rapid diagnosis and typind of herpesvirus hominis by the fluorescent antibody method.Scandinavian journal of infectious diseases, 6 2
A. Spriggs (1966)
CytodiagnosisBritish Medical Journal, 2
J. Kerr (1971)
Shrinkage necrosis: A distinct mode of cellular deathThe Journal of Pathology, 105
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Dynamics of growth in tumors and in normal organisms.National Cancer Institute monograph, 30
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Direct 1mmunofluorescen1 test for the diagno,i, of genital herpes virus infections
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Ultrastructural localization of aryl sulfatase in human epidermis.Acta dermato-venereologica, 48 6
E. Malaise, M. Tubiana (1968)
Cell proliferation kinetics in five human solid tumorsCancer, 22
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Perinatal risk associated with maternal genital herpes simplex virus infection.American journal of obstetrics and gynecology, 110 6
Yoshinori Kawagoe (1948)
Pathology of TumoursNature, 162
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Some lysosome functions in liver cells reacting to sublethal injury
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Cell loss in malignant tumours in man.European journal of cancer, 3 3
Departments of Pathology, University of Queensland Medical School and Royal Brisbane Hospital, Herston, Brisbane, Australia PLATES XXII AND XXIII THEhigh incidence of mitotic figures in slowly growing basal-cell carcinomas of skin has long been an enigma (Willis, 1953), and recent detailed studies of cell proliferation in these tumours have confirmed that there is indeed a gross discrepancy between their expected and observed rate of enlargement (Frindel, Malaise and Tubiana, 1968; Weinstein and Frost, 1970). It has been suggested that extensive and continuous loss of neoplastic cells is the most likely explanation for this discrepancy, but the actual mechanism of the loss has remained obscure. Progressive loss of hepatocytes due to a distinct type of cellular death termed shrinkage necrosis (Kerr, 1965, 1971) has been shown to be largely responsible for the rapid atrophy of rat liver tissue that follows obstruction of its portal blood supply (Kerr, 1971, 1972). This process characteristically affects scattered individual cells, which initially condense and separate from their neighbours. Numerous buds then develop on their surfaces and subsequently separate to produce small rounded masses of compacted but well-preserved cytoplasm, which may or may not contain pyknotic nuclear remnants. Such structures, customarily referred to as
The Journal of Pathology – Wiley
Published: May 1, 1972
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