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J. Moore, H. Garner, J. Coffman (1981)
Haematological changes during development of acute laminitis hypertension.Equine veterinary journal, 13 4
H. Garner, J. Moore, Jerry Johnson, L. Clark, J. Amend, Tritschler Lg, J. Coffmann, R. Sprouse, D. Hutcheson, C. Salem (1978)
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L. Clarke, H. Garner, D. Hatfield (1982)
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H. Garner, A. Hahn, C. Salem, J. Coffman, D. Hutcheson, J. Johnson (1977)
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Hood (1982)
The use of alpha and beta adrenergic blockade as a preventative in the carbohydrate model of laminitis: A preliminary report; heparin as a preventative for equine laminitis: A preliminary report; The effect of exogenous steroid on the carbohydrate overload model of equine laminitis: A preliminary reportProceedings of First Equine Endotoxemia-Laminitis Symposium, 1
Summary Thirteen (65 per cent) of 20 horses subjected to carbohydrate overload developed Obel Grade 3 lameness within 56 h. Increases in plasma endotoxin from control levels of less than 0.1 ng/litre to values ranging from 2.4 to 81.53 ng/litre were measured in 11 (85 per cent) of 13 horses during the onset of Obel Grade 3 lameness. Obel Grade 3 lameness was associated with rises in plasma Gram‐negative endotoxin levels in 11 (92 per cent) of 12 horses. Two peak increases separated by 16 h were verified in five (45 per cent) of 11 horses that exhibited both endotoxaemia and Obel Grade 3 lameness. The first peak occurred, on average, at 32 h and (he second peak at 48 h post overload when only one peak was measured, this occurred in four horses at the average time of 24 h whereas in another three horses only a 48 h or 56 h peak was detected after carbohydrate overload.
Equine Veterinary Journal – Wiley
Published: Jan 1, 1987
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