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The Epidemiology and Control of Open Angle Glaucoma: A Population-Based Perspective

The Epidemiology and Control of Open Angle Glaucoma: A Population-Based Perspective Chronic open angle glaucoma is an etiologically heterogeneous group of diseases characterized by damage to the optic nerve, resulting in peripheral visual loss that can progress to involve the fovea and central vision. Open angle glaucoma can be divided into primary conditions and conditions which are secondary to another ocular or systemic disease. Causes of secondary glaucoma include uveitis, cataract, trauma, and disorders affecting the development structure of the angle. This review focuses on primary open angle glaucoma (POAG), since it accounts for the vast majority of the disease burden in the US population and its etiology remains unknown. http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Annual Review of Public Health Annual Reviews

The Epidemiology and Control of Open Angle Glaucoma: A Population-Based Perspective

Annual Review of Public Health , Volume 17 (1) – May 1, 1996

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Publisher
Annual Reviews
Copyright
Copyright 1996 Annual Reviews. All rights reserved
Subject
Review Articles
ISSN
0163-7525
eISSN
1545-2093
DOI
10.1146/annurev.pu.17.050196.001005
pmid
8724220
Publisher site
See Article on Publisher Site

Abstract

Chronic open angle glaucoma is an etiologically heterogeneous group of diseases characterized by damage to the optic nerve, resulting in peripheral visual loss that can progress to involve the fovea and central vision. Open angle glaucoma can be divided into primary conditions and conditions which are secondary to another ocular or systemic disease. Causes of secondary glaucoma include uveitis, cataract, trauma, and disorders affecting the development structure of the angle. This review focuses on primary open angle glaucoma (POAG), since it accounts for the vast majority of the disease burden in the US population and its etiology remains unknown.

Journal

Annual Review of Public HealthAnnual Reviews

Published: May 1, 1996

There are no references for this article.