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C. Pollitt (1994)
The basement membrane at the equine hoof dermal epidermal junction.Equine veterinary journal, 26 5
Philip Johnson, S. Tyagi, L. Katwa, V. Ganjam, L. Moore, J. Kreeger, N. Messer (1998)
Activation of extracellular matrix metalloproteinases in equine laminitisVeterinary Record, 142
C. Pollitt (1996)
Basement membrane pathology: a feature of acute equine laminitis.Equine veterinary journal, 28 1
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Ultrastructure of the equine hoof wall secondary epidermal lamellae.American Journal of Veterinary Research, 44
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Structure and function of hemidesmosomes: more than simple adhesion complexes.The Journal of investigative dermatology, 112 4
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Reduced epithelial adhesion after extended contact lens wear correlates with reduced hemidesmosome density in cat cornea.Investigative ophthalmology & visual science, 33 2
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The dermal-epidermal junction.Current opinion in cell biology, 9 5
Pollitt (1998)
Batimastat (BB94) inhibits matrix metalloproteinases of equine laminitisEquine vet. J., 26
K. French, C. Pollitt (2010)
Equine laminitis: cleavage of laminin 5 associated with basement membrane dysadhesion.Equine veterinary journal, 36 3
M. Kyaw-Tanner, C. Pollitt (2010)
Equine laminitis: increased transcription of matrix metalloproteinase-2 (MMP-2) occurs during the developmental phase.Equine veterinary journal, 36 3
B. Mungall, C. Pollitt, R. Collins (1998)
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Decreased glucose metabolism causes separation of hoof lamellae in vitro:could this be a trigger for laminitis?Equine vet. J., 26
Reasons for performing study: Light microscopical studies show that the key lesion of laminitis is separation at the hoof lamellar dermal‐epidermal interface. More precise knowledge of the damage occurring in the lamellar basement membrane zone may result if laminitis affected tissue is examined with the transmission electron microscope. This could lead to better understanding of the pathogenesis of lesions and the means of treatment or prevention.
Equine Veterinary Journal – Wiley
Published: Jan 1, 2004
Keywords: ; ; ; ; ;
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