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S. Kajigaya, T. Shimada, S. Fujita, N. Young (1989)
A genetically engineered cell line that produces empty capsids of B19 (human) parvovirus.Proceedings of the National Academy of Sciences of the United States of America, 86 19
M. Söderlund-Venermo, K. Brown, D. Erdman (1987)
[Human parvoviruses].Annales de pediatrie, 34 7
Copland Jg, Davies Ct (1964)
SPREAD OF VIRUS HEPATITIS.The Lancet, 1
J. Clewley (1989)
Polymerase chain reaction assay of parvovirus B19 DNA in clinical specimensJournal of Clinical Microbiology, 27
G. Kurtzman, P. Meyers, B. Cohen, Adoor Amunullah, N. Young (1988)
PERSISTENT B19 PARVOVIRUS INFECTION AS A CAUSE OF SEVERE CHRONIC ANAEMIA IN CHILDREN WITH ACUTE LYMPHOCYTIC LEUKAEMIAThe Lancet, 332
S. Hall, B. Cohen, P. Mortimer, E. Caul, J. Cradock-Watson, M. Anderson, J. Pattison, J. Shirley, T. Peto (1991)
Prospective study of human parvovirus (B19) infection in pregnancyInternational Journal of Gynecology & Obstetrics, 34
P. Juto, B. Settergren (1988)
Specific serum IgA, IgG and IgM antibody determination by a modified indirect ELISA-technique in primary and recurrent herpes simplex virus infection.Journal of virological methods, 20 1
M. Anderson, M. Anderson, S. Jones, A. Minson (1985)
Diagnosis of human parvovirus infection by dot‐blot hybridization using cloned viral DNAJournal of Medical Virology, 15
T. Schwarz, Michael Roggendorf, F. Deinhardt (1988)
Human parvovirus B19: ELISA and immunoblot assays.Journal of virological methods, 20 2
Anderson Anderson (1990)
Human parvovirusesJournal of Infectious Diseases, 161
Yaegashi Nobuo, Shiraishi Hiroyuki, Tadaki Kohtaro, Yajima Akira, Sugamura Kazuo (1989)
Enzyme-linked immunosorbent assay for IgG and IgM antibodies against human parvovirus B19: use of monoclonal antibodies and viral antigen propagated in vitro.Journal of virological methods, 26 2
W. Koch, S. Adler (1990)
Detection of human parvovirus B19 DNA by using the polymerase chain reactionJournal of Clinical Microbiology, 28
R. Shade, M. Blundell, S. Cotmore, P. Tattersall, C. Astell (1986)
Nucleotide sequence and genome organization of human parvovirus B19 isolated from the serum of a child during aplastic crisisJournal of Virology, 58
Hall Hall, Cohen Cohen, Mortimer Mortimer, Shirley Shirley, Peto Peto (1990)
Prospective study of human parvovirus (B19) infection in pregnancyBritish Medical Journal, 300
M. Salimans, F. Rijke, A. Raap, A. Elsacker-Niele (1989)
Detection of parvovirus B19 DNA in fetal tissues by in situ hybridisation and polymerase chain reaction.Journal of Clinical Pathology, 42
B. Cohen, P. Mortimer, M. Pereira (1983)
Diagnostic assays with monoclonal antibodies for the human serum parvovirus-like virus (SPLV)Journal of Hygiene, 91
D. Reid, T. Brown, T. Reid, J. Rennie, C. Eastmond (1985)
HUMAN PARVOVIRUS-ASSOCIATED ARTHRITIS: A CLINICAL AND LABORATORY DESCRIPTIONThe Lancet, 325
M. Anderson, P. Higgins, L. Davis, J. Willman, S. Jones, I. Kidd, J. Pattison, D. Tyrrell (1985)
Experimental parvoviral infection in humans.The Journal of infectious diseases, 152 2
K. Brown, M. Buckley, B. Cohen, D. Samuel (1989)
An amplified ELISA for the detection of parvovirus B19 IgM using monoclonal antibody to FITC.Journal of virological methods, 26 2
G. Kurtzman, N. Frickhofen, J. Kimball, D. Jenkins, A. Nienhuis, N. Young (1989)
Pure red-cell aplasia of 10 years' duration due to persistent parvovirus B19 infection and its cure with immunoglobulin therapy.The New England journal of medicine, 321 8
(1988)
Intrauterine infection with human parvovirus B19: a light and electron microscopy study
N. Frickhofen, J. Abkowitz, M. Safford, J. Berry, J. Antunez-de-Mayolo, A. Astrow, Robert Cohen, I. Halperin, L. King, D. Mintzer, B. Cohen, N. Young (1990)
Persistent B19 parvovirus infection in patients infected with human immunodeficiency virus type 1 (HIV-1): a treatable cause of anemia in AIDS.Annals of internal medicine, 113 12
Larry Anderson, C. Tsou, Robert Parker, Terence Chorba, H. Wulff, P. Tattersall, P. Mortimer (1986)
Detection of antibodies and antigens of human parvovirus B19 by enzyme-linked immunosorbent assayJournal of Clinical Microbiology, 24
Terence Chorba, P. Coccia, R. Holman, P. Tattersall, Larry Anderson, J. Sudman, N. Young, E. Kurczynski, U. Saarinen, Robyn Moir, D. Lawrence, J. Jason, B. Evatt (1986)
The role of parvovirus B19 in aplastic crisis and erythema infectiosum (fifth disease).The Journal of infectious diseases, 154 3
(1989)
Risks associated with human parvovirus B19 infection.MMWR. Morbidity and mortality weekly report, 38 6
B. Cohen, M. Buckley, J. Clewley, V. Jones, A. Puttick, R. Jacoby (1986)
Human parvovirus infection in early rheumatoid and inflammatory arthritis.Annals of the Rheumatic Diseases, 45
To determine the diagnostic use of different markers of acute parvovirus 619 infection, serum specimens obtained from 128 persons with erythema infectiosum were tested for specific immunoglobulin G (IgG), IgA, and IgM antibodies by capture enzyme immunoassay (EIA) using Chinese hamster ovary (CHO) cell‐expressed B19 antigen, and tested for circulating B19 DNA by polymerase chain reaction (PCR). A significant rise in specific IgG and IgA antibodies was detected in 87% and 77%, respectively, of persons from whom acute‐ and convalescent‐phase serum specimens were available. Specific IgA antibodies were detected in single serum specimens from 90% of cases and were present in 22 (18%) of 120 persons from a control group without a history of recent exposure to B19. Specific IgM antibodies were detected i n 97% of cases and one person (IYO) from the control group. B19 DNA was detected in 94% of cases and was absent in 20 persons from the control group positive for both IgG and IgA antibodies. Serum specimens obtained between 4 and 6 months after onset of illness from six additional persons were also tested. All had specific IgG antibodies, four (67%) had IgA, five (83%) had IgM, and none had detectable B19 DNA. Our data indicate that 1) specific IgA antibodies are too persistent to be a useful indicator of recent B19 infection; 2) specific IgM antibodies are the most sensitive indicator of acute B19 infection in immunologically normal persons but can persist up to 6 months; and 3) 619 DNA can often be detected up to 2 months after onset of illness even in immunologically normal hosts and might be a useful adjunct test for diagnosis of acute B19 infection.
Journal of Medical Virology – Wiley
Published: Oct 1, 1991
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