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' , ; SATELLITE ARTICLE See page I J 9 for correspondinn Case Remt Diagnosis of hyperadrenocorticism in the horse M. H. HILLYER, F. G. R. TAYLOR, T. S. MAIR", D. MURPHY?, T. D. G. WATSON? and S. LOWt Departments of Veterinary Medicine and Surgery, University of Bristol School of Veterinary Science, Langford House, Langford, Bristol BSl8 7DU, UK; * I 23 Commercial Road, Paddock Wood, Tonbridge, Kent TN12 6DS, UK; and ?Department of Veterinary Medicine, University of Glasgow Veterinary School, Bearsden Road, Bearsden, Glasgow G61 1 QH, UK. INTRODUCTION Hyperadrenocorticism (HAC) in the horse, although recognised for many years (Loeb, Capen and Johnson 1966), has recently been reported increasingly (Orth and Nicholson 1982; Allen, Barbee and Crisman 1988; Horvath, Ames, Metz and Larson 1988; Staempfli, Eigenmann and Clarke 1988; see Beck p 119). HACresults primarily from an adenoma of the intermediate lobe of the pituitary gland which produces n excess secretions, including ACTH, which i turn cause adrenocortical hypertrophy (Moore, Steiss, Nicholson and Orth 1979). Although a primary adrenal cortical adenocarcinoma has been reported (Raker and Fegley 1965), diagnosis of HAC generally depends on demonstrating the presence and/or effects of a functional pituitary adenoma with secondary adrenocortical
Equine Veterinary Education – Wiley
Published: Jun 1, 1992
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