Introduction Vitamin A is essential for the differentiation and function of normal tissue and repair after injury. Recent studies have reported an association between vitamin A deficiency and chronic lung disease (CLD) in preterm infants [7, 11]. Two studies have demonstrated that infants with chronic lung disease require prolonged intravenous nutrition [7, 11]. It is therefore tempting to speculate that poor supplementation, while such infants remain nil by mouth, is the most likely explanation for the association of vitamin A deficiency and CLD. Although this hypothesis was supported by data from one study , it was contradicted by a second  which suggested infants with CLD actually receive more vitamin A than those without CLD . HUSTEAD et al  suggested other mechanisms than inadequate administration of vitamin A should be considered to explain the association of CLD and low vitamin A levels; such as lack of mobilisation of retinol from hepatic stores, higher needs or more rapid utilisation of vitamin A in infants with CLD. If those suggestions are correct , infants at risk of developing CLD would require higher levels of vitamin A supplementation. Vitamin A supplementation, however, may be hazardous, as toxic levels result in
Journal of Perinatal Medicine – de Gruyter
Published: Jan 1, 1993
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