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H. Lippman (1924)
A MORPHOLOGIC AND QUANTITATIVE STUDY OF THE BLOOD CORPUSCLES IN THE NEW-BORN PERIODJAMA Pediatrics, 27
P. McMaster (1924)
STUDIES ON THE TOTAL BILE VI. THE INFLUENCE OF DIET UPON THE OUTPUT OF CHOLESTEROL IN THE BILE.Journal of Experimental Medicine, 40
W. Lucas, Bradford Dearing, Hal Hoobler, A. Cox, Martha Jones, F. Smyth (1921)
BLOOD STUDIES IN THE NEW-BORN: MORPHOLOGICAL; CHEMICAL; COAGULATION; UROBILIN AND BILIRUBINJAMA Pediatrics, 22
G. Whiffle, C. Hooper (1913)
HEMATOGENOUS AND OBSTRUCTIVE ICTERUSThe Journal of Experimental Medicine, 17
B. Jones (1921)
ISO-AGGLUTININS IN THE BLOOD OF THE NEW-BORNJAMA Pediatrics, 22
J. Aitken (1902)
BLOOD‐COUNTS IN THE NEW‐BORN.BJOG: An International Journal of Obstetrics & Gynaecology, 1
F. Mann, J. Bollman, T. Magath (1924)
STUDIES ON THE PHYSIOLOGY OF THE LIVER: IX. The Formation of Bile Pigment after Total Removal of the LiverAmerican Journal of Physiology, 69
P. McMaster, G. Broun, P. Rous (1923)
STUDIES ON THE TOTAL BILE : I. THE EFFECTS OF OPERATION, EXERCISE, HOT WEATHER, RELIEF OF OBSTRUCTION, INTERCURRENT DISEASE, AND OTHER NORMAL AND PATHOLOGICAL INFLUENCES.Journal of Experimental Medicine, 37
G. Whiffle, C. Hoofer (1913)
ICTERUS : A RAPID CHANGE OF HEMOGLOBIN TO BILE PIGMENT IN THE CIRCULATION OUTSIDE THE LIVER.The Journal of experimental medicine, 17 6
A. Fehrsen (1903)
The hæmoglobin and corpuscular content of the blood of the newbornThe Journal of Physiology, 30
C. Grulee, A. Mebane (1925)
THE VAN DEN BERGH TEST FOR ICTERUS IN THE BLOOD OF INFANTSJAMA Pediatrics, 30
E. Ravdin (1925)
AN ESTIMATION OF THE CLINICAL VALUE OF THE VAN DEN BERGH TESTThe American Journal of the Medical Sciences, 169
H. Bourquin (1922)
A STUDY ON THE PERMEABILITY OF THE PLACENTA: I. Permeability to Agglutinins, Hemolysins, Diphtheria Antitoxin and DiastaseAmerican Journal of Physiology, 59
P. Klemperer (1924)
ICTERUS GRAVIS OF THE NEW-BORNJAMA Pediatrics, 28
A. Bergh, I. Snapper, J. Muller
Der Gallenfarbstoff im Blute
A. Hess (1912)
A STUDY OF ICTERUS NEONATORUM BY MEANS OF THE DUODENAL CATHETERJAMA Pediatrics
In 1882, Hofmeier1 first suggested a hematogenous origin for jaundice of the new-born infant. For years this theory received little attention, but evidence gradually accumulated which led writers on the subject to grant it a rôle of minor importance, then to assign it a parity with hepatogenous factors, and finally to advance it as the sole cause of this condition. In 1912, Hess2 dismissed blood destruction as being a primary cause of jaundice, on the following grounds: First, when the liver is extirpated, jaundice cannot be induced; second, the finding of bile acids in the pericardial fluid of jaundiced babies proves the participation of the liver; third, free hemoglobin cannot be found in the blood of the new-born infant; fourth, jaundice cannot be produced by the injection of hemoglobin, and fifth, jaundice does not follow transfusion. Today each of these objections can be met. Whipple and Hooper3
American journal of diseases of children – American Medical Association
Published: Sep 1, 1928
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