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Hyperparathyroidism

Hyperparathyroidism This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables. Abstract This monograph presents, in repetitious detail, the unorthodox attitudes of the authors regarding the clinical and pathologic characteristics of primary hyperparathyroidism and its surgical treatment. The scientific facts on which these attitudes are based are qualitatively accurate, but the quantitative conclusions appear to be exaggerated in many instances. As examples, statements claiming a rarity today of classic cases of parathyroid adenoma; frequent hypercalcemia in Hashimoto thyroiditis due to loss of calcitonin secretory activity; large supplies of calcitonin in the adrenal medulla; the association of sprains, varicose veins, hernias, hemorrhoids, clubbing, and congestive heart failure with primary hyperparathyroidism; a significant frequency of normocalcemic hyperparathyroidism; hypercalcemia in Cushing syndrome; and a "destiny" for patients with idiopathic hypoparathyroidism to develop Addison disease and/or pernicious anemia all go far beyond the actual facts. Further, the authors insist that a major action of parathyroid hormone is to produce adverse catabolic effects in all connective tissues http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Archives of Surgery American Medical Association

Hyperparathyroidism

Archives of Surgery , Volume 109 (4) – Oct 1, 1974

Hyperparathyroidism

Abstract

This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables. Abstract This monograph presents, in repetitious detail, the unorthodox attitudes of the authors regarding the clinical and pathologic characteristics of primary hyperparathyroidism and its surgical treatment. The scientific facts on which these attitudes are based are qualitatively accurate, but the quantitative conclusions appear to be exaggerated in many...
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Publisher
American Medical Association
Copyright
Copyright © 1974 American Medical Association. All Rights Reserved.
ISSN
0004-0010
eISSN
1538-3644
DOI
10.1001/archsurg.1974.01360040099038
Publisher site
See Article on Publisher Site

Abstract

This article is only available in the PDF format. Download the PDF to view the article, as well as its associated figures and tables. Abstract This monograph presents, in repetitious detail, the unorthodox attitudes of the authors regarding the clinical and pathologic characteristics of primary hyperparathyroidism and its surgical treatment. The scientific facts on which these attitudes are based are qualitatively accurate, but the quantitative conclusions appear to be exaggerated in many instances. As examples, statements claiming a rarity today of classic cases of parathyroid adenoma; frequent hypercalcemia in Hashimoto thyroiditis due to loss of calcitonin secretory activity; large supplies of calcitonin in the adrenal medulla; the association of sprains, varicose veins, hernias, hemorrhoids, clubbing, and congestive heart failure with primary hyperparathyroidism; a significant frequency of normocalcemic hyperparathyroidism; hypercalcemia in Cushing syndrome; and a "destiny" for patients with idiopathic hypoparathyroidism to develop Addison disease and/or pernicious anemia all go far beyond the actual facts. Further, the authors insist that a major action of parathyroid hormone is to produce adverse catabolic effects in all connective tissues

Journal

Archives of SurgeryAmerican Medical Association

Published: Oct 1, 1974

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