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Eight-and-a-Half Syndrome

Eight-and-a-Half Syndrome Clinical Review & Education Images in Neurology Jone Bocos-Portillo, MD; Javier Ruiz Ojeda, MD; Marian Gomez-Beldarrain, MD; Raquel Vazquez-Picon, MD; Juan Carlos Garcia-Monco, MD An 81-year-old patient was seen with sudden onset of ophthal- and-a-half syndrome, first described in 1998 by Eggenberger. moplegia and unsteadiness. His medical record included hyperten- This syndrome is caused by a lesion, most often vascular or demy- sion, type 2 diabetes mellitus, and hypercholesterolemia. The neu- elinating, in the dorsal tegmentum of the caudal pons involving rological examination disclosed the parapontine reticular formation and the medial longitudinal left internuclear ophthalmople- fasciculus, as well as the nucleus and the fasciculus of the facial Video at jamaneurology.com gia and horizontal left-sided nerve. gaze palsy, accompanied by left-sided peripheral facial palsy Figure, A shows the lesion location at the pons level. The pa- (Video). The physical examination findings were otherwise normal. tient’s cranial magnetic resonance imaging revealed an ischemic in- farct involving the pons (Figure, B-C). Discussion Progressive improvement was noted during follow-up of the The combination of one-and-a-half syndrome plus an ipsilateral patient. At 2 months after admission, he demonstrated resolution facial nerve palsy (lower motor neuron type) constitutes eight- of ophthalmoplegia and persistence of mild http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png JAMA Neurology American Medical Association

Eight-and-a-Half Syndrome

Abstract

Clinical Review & Education Images in Neurology Jone Bocos-Portillo, MD; Javier Ruiz Ojeda, MD; Marian Gomez-Beldarrain, MD; Raquel Vazquez-Picon, MD; Juan Carlos Garcia-Monco, MD An 81-year-old patient was seen with sudden onset of ophthal- and-a-half syndrome, first described in 1998 by Eggenberger. moplegia and unsteadiness. His medical record included hyperten- This syndrome is caused by a lesion, most often vascular or demy- sion, type 2 diabetes mellitus, and hypercholesterolemia....
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Publisher
American Medical Association
Copyright
Copyright 2015 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.
ISSN
2168-6149
eISSN
2168-6157
DOI
10.1001/jamaneurol.2015.0255
pmid
25962103
Publisher site
See Article on Publisher Site

Abstract

Clinical Review & Education Images in Neurology Jone Bocos-Portillo, MD; Javier Ruiz Ojeda, MD; Marian Gomez-Beldarrain, MD; Raquel Vazquez-Picon, MD; Juan Carlos Garcia-Monco, MD An 81-year-old patient was seen with sudden onset of ophthal- and-a-half syndrome, first described in 1998 by Eggenberger. moplegia and unsteadiness. His medical record included hyperten- This syndrome is caused by a lesion, most often vascular or demy- sion, type 2 diabetes mellitus, and hypercholesterolemia. The neu- elinating, in the dorsal tegmentum of the caudal pons involving rological examination disclosed the parapontine reticular formation and the medial longitudinal left internuclear ophthalmople- fasciculus, as well as the nucleus and the fasciculus of the facial Video at jamaneurology.com gia and horizontal left-sided nerve. gaze palsy, accompanied by left-sided peripheral facial palsy Figure, A shows the lesion location at the pons level. The pa- (Video). The physical examination findings were otherwise normal. tient’s cranial magnetic resonance imaging revealed an ischemic in- farct involving the pons (Figure, B-C). Discussion Progressive improvement was noted during follow-up of the The combination of one-and-a-half syndrome plus an ipsilateral patient. At 2 months after admission, he demonstrated resolution facial nerve palsy (lower motor neuron type) constitutes eight- of ophthalmoplegia and persistence of mild

Journal

JAMA NeurologyAmerican Medical Association

Published: Jul 1, 2015

References

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