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Connecting the "Dots" of Brain Dysfunction in Schizophrenia

Connecting the "Dots" of Brain Dysfunction in Schizophrenia COMMENTARY Connecting the “Dots” of Brain Dysfunction in Schizophrenia What Does the Picture Look Like? N THIS issue of the lying) process(es) that produce the symptomatic functioning between ARCHIVES, Dr Nancy An- outward appearance that we call these episodes? Is it reasonable to cat- dreasen proposes a “uni- schizophrenia. She supports the well- egorize both of these patients in the tary” neural circuit–cogni- documented argument that, in ex- same diagnostic “bin” called schizo- I tive dysfunction model for ploring the “genetics of schizophre- phrenia? Is it likely that we will find understanding schizophrenia. This nia,” we should rely on “core” a common neural and cognitive dys- commentary will review Andreas- cognitive dysfunctions (ie, interme- function for these seemingly di- en’s scholarly article with a focus on diate phenotypic markers) rather verse states? I believe the odds are (1) the continued search for specific that we will not, because the disor- than on less precise diagnostic crite- 2-4 neural circuit abnormalities in schizo- der is truly heterogeneous, as de- ria. Andreasen notes that on the phrenia and their associated “signa- fined by DSM-IV and by clinicians surface schizophrenia seems to be ture” cognitive deficits; (2) how these and researchers. Unfortunately, http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png JAMA Psychiatry American Medical Association

Connecting the "Dots" of Brain Dysfunction in Schizophrenia

JAMA Psychiatry , Volume 56 (9) – Sep 1, 1999

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Publisher
American Medical Association
Copyright
Copyright 1999 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.
ISSN
2168-622X
eISSN
2168-6238
DOI
10.1001/archpsyc.56.9.791
Publisher site
See Article on Publisher Site

Abstract

COMMENTARY Connecting the “Dots” of Brain Dysfunction in Schizophrenia What Does the Picture Look Like? N THIS issue of the lying) process(es) that produce the symptomatic functioning between ARCHIVES, Dr Nancy An- outward appearance that we call these episodes? Is it reasonable to cat- dreasen proposes a “uni- schizophrenia. She supports the well- egorize both of these patients in the tary” neural circuit–cogni- documented argument that, in ex- same diagnostic “bin” called schizo- I tive dysfunction model for ploring the “genetics of schizophre- phrenia? Is it likely that we will find understanding schizophrenia. This nia,” we should rely on “core” a common neural and cognitive dys- commentary will review Andreas- cognitive dysfunctions (ie, interme- function for these seemingly di- en’s scholarly article with a focus on diate phenotypic markers) rather verse states? I believe the odds are (1) the continued search for specific that we will not, because the disor- than on less precise diagnostic crite- 2-4 neural circuit abnormalities in schizo- der is truly heterogeneous, as de- ria. Andreasen notes that on the phrenia and their associated “signa- fined by DSM-IV and by clinicians surface schizophrenia seems to be ture” cognitive deficits; (2) how these and researchers. Unfortunately,

Journal

JAMA PsychiatryAmerican Medical Association

Published: Sep 1, 1999

References

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