Access the full text.
Sign up today, get DeepDyve free for 14 days.
References for this paper are not available at this time. We will be adding them shortly, thank you for your patience.
Will further study of our reptilian kindred (whose hearts demonstrate a unique anaerobic metabolic capacity) yield a treatment for myocardial infarction? Possibly, suggests one of the contributors to Clinical Cardiovascular Physiology. He cites many approaches to the problem of inadequate myocardial oxygenation, including experimental attempts to increase glycolytic flux, enhance excitation-contraction coupling, and improve coronary blood flow. Unfortunately, methods that can effectively increase the energy output of the ischemic myocardial cell have yet to be developed. However, reducing the work load of the ischemic heart (with propranolol, furosamide, or nitroglycerine and nitroprusside) helps balance energy demand and supply. Although they consider the role of the digitalis glucosides and other treatment, Dr Levine and the 39 contributors to his book mostly present their concepts of normal and abnormal cardiac physiology. They also take up the effects of disordered cardiac function on the kidneys and lungs, often in much detail. Schreiner and
JAMA – American Medical Association
Published: Nov 1, 1976
Read and print from thousands of top scholarly journals.
Already have an account? Log in
Bookmark this article. You can see your Bookmarks on your DeepDyve Library.
To save an article, log in first, or sign up for a DeepDyve account if you don’t already have one.
Copy and paste the desired citation format or use the link below to download a file formatted for EndNote
Access the full text.
Sign up today, get DeepDyve free for 14 days.
All DeepDyve websites use cookies to improve your online experience. They were placed on your computer when you launched this website. You can change your cookie settings through your browser.