Get 20M+ Full-Text Papers For Less Than $1.50/day. Start a 14-Day Trial for You or Your Team.

Learn More →

Clinical Cardiovascular Physiology

Clinical Cardiovascular Physiology Will further study of our reptilian kindred (whose hearts demonstrate a unique anaerobic metabolic capacity) yield a treatment for myocardial infarction? Possibly, suggests one of the contributors to Clinical Cardiovascular Physiology. He cites many approaches to the problem of inadequate myocardial oxygenation, including experimental attempts to increase glycolytic flux, enhance excitation-contraction coupling, and improve coronary blood flow. Unfortunately, methods that can effectively increase the energy output of the ischemic myocardial cell have yet to be developed. However, reducing the work load of the ischemic heart (with propranolol, furosamide, or nitroglycerine and nitroprusside) helps balance energy demand and supply. Although they consider the role of the digitalis glucosides and other treatment, Dr Levine and the 39 contributors to his book mostly present their concepts of normal and abnormal cardiac physiology. They also take up the effects of disordered cardiac function on the kidneys and lungs, often in much detail. Schreiner and http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png JAMA American Medical Association

Clinical Cardiovascular Physiology

JAMA , Volume 236 (18) – Nov 1, 1976

Loading next page...
 
/lp/american-medical-association/clinical-cardiovascular-physiology-1UWf0Xm9Zv

References (0)

References for this paper are not available at this time. We will be adding them shortly, thank you for your patience.

Publisher
American Medical Association
Copyright
Copyright © 1976 American Medical Association. All Rights Reserved. Applicable FARS/DFARS Restrictions Apply to Government Use.
ISSN
0098-7484
eISSN
1538-3598
DOI
10.1001/jama.1976.03270190064045
Publisher site
See Article on Publisher Site

Abstract

Will further study of our reptilian kindred (whose hearts demonstrate a unique anaerobic metabolic capacity) yield a treatment for myocardial infarction? Possibly, suggests one of the contributors to Clinical Cardiovascular Physiology. He cites many approaches to the problem of inadequate myocardial oxygenation, including experimental attempts to increase glycolytic flux, enhance excitation-contraction coupling, and improve coronary blood flow. Unfortunately, methods that can effectively increase the energy output of the ischemic myocardial cell have yet to be developed. However, reducing the work load of the ischemic heart (with propranolol, furosamide, or nitroglycerine and nitroprusside) helps balance energy demand and supply. Although they consider the role of the digitalis glucosides and other treatment, Dr Levine and the 39 contributors to his book mostly present their concepts of normal and abnormal cardiac physiology. They also take up the effects of disordered cardiac function on the kidneys and lungs, often in much detail. Schreiner and

Journal

JAMAAmerican Medical Association

Published: Nov 1, 1976

There are no references for this article.