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Ingalls Th, Aycock Wl (1951)
Upper-respiratory infection as a factor influencing susceptibility to poliomyelitis.The New England Journal of Medicine, 245
F. Top (1952)
Occurrence of poliomyelitis in relation to tonsillectomies at various intervals.Journal of the American Medical Association, 150 6
D. Cunning (1948)
Tonsillectomy‐poliomyelitis survey —1947The Laryngoscope, 58
F. Top, H. Vaughan (1941)
Epidemiology of Poliomyelitis in Detroit in 1939.American journal of public health and the nation's health, 31 8
G. Anderson, G. Anderson, A. Skaar, F. Sandler (1950)
LVI The Risk of Poliomyelitis after TonsillectomyAnnals of Otology, Rhinology & Laryngology, 59
W. Aycock (1942)
TONSILLECTOMY AND POLIOMYELITIS: I. EPIDEMIOLOGIC CONSIDERATIONSMedicine, 20
John Anderson (1945)
Poliomyelitis and recent tonsillectomyThe Journal of Pediatrics, 27
D. Cunning (1947)
Tonsillectomy and poliomyelitis.Archives of otolaryngology, 46 5
W. Aycock, E. Luther (1929)
The Occurrence of Poliomyelitis Following TonsillectomyThe New England Journal of Medicine, 200
Kinney Ce (1948)
Tonsillectomy and poliomyelitis in Cuyahoga County, Ohio.The Ohio State medical journal, 44
D. Bodian (1952)
A reconsideration of the pathogenesis of poliomyelitis.American journal of hygiene, 55 3
D. Cunning (1949)
Poliomyelitis‐tonsillectomy survey‐1948The Laryngoscope, 59
A. Fischer, M. Stillerman, H. Marks (1941)
RELATION OF TONSILLECTOMY AND OF ADENOIDECTOMY TO THE INCIDENCE OF POLIOMYELITIS: WITH SPECIAL REFERENCE TO THE BULBAR FORMJAMA Pediatrics, 61
L. Weinstein, M. Vogel, N. Weinstein (1954)
A study of the relationship of the absence of tonsils to the incidence of bulbar poliomyelitis.The Journal of pediatrics, 44 1
R. Eley, C. Flake (1938)
Acute anterior poliomyelitis following tonsillectomy and adenoidectomyThe Journal of Pediatrics, 13
F. Maccallum (1955)
The Pathogenesis of PoliomyelitisJournal of Clinical Pathology, 8
Southcott Rv (1953)
Studies on a long range association between bulbar poliomyelitis and previous tonsillectomy.The Medical Journal of Australia, 2
Foremost among the many unknowns in the pathogenesis of poliomyelitis are the factors that determine the type of response to the infection. There is ample evidence that infection with the poliomyelitis viruses is widespread, ultimately involving almost all persons, except those in isolated areas. In most of these persons, the infection is mild, producing no paralysis and usually occuring without recognizable symptoms. Yet a small number of persons respond badly to the virus invasion, with resultant paralysis. Of those with central nervous system involvement, a few show involvement of the vital medullary centers, while the majority show lesions of the spinal cord. The proportion with bulbar paralysis has supposedly increased in recent years and is greater in the older age groups. Very little evidence is currently available, however, as to the factors that determine this localization of the paralysis in the bulbar area. Among the many factors that have been
JAMA – American Medical Association
Published: Jul 24, 1954
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